Cells build carbohydrate polymers by using energy to form glycosidic linkages, the bonds between monosaccharides. A dehydration synthesis reaction forms a bond between carbon atoms in two monosaccharides, sandwiching an oxygen atom between them and releasing a water molecule. Hope this helps!
Answer: diabetes mellitus
Explanation:
This is a disease condition associated with elevated blood glucose levels.
It is due to abnormalities of the Pancreas. The pancreas coordinates two hormones INSULIN AND GLUCAGON in its cells.
INSULIN converts excess sugar to GLYCOGEN while GLUCAGON breaks down GLYCOGEN to sugar when blood glucose/glucose levels drops.
If the pancreas beta-cells of glucose are faulty, INSULIN fails to converts excess glucose to glycogen; therefore glucose builds up in the blood plasma,and failed to enter the needed cells. Large amount of glucose is wasted in urine.This is diabetes Mellitus.
The two types of diabetes are:
Type 1 and Type 2.
Answer: protein > neurons > cardiac muscle > bladder > digestive system > human being
Explanation:
protein molecule, neurons individual cells, muscle tissue, bladder organ, digestive system interconnected organs, human being whole body
Answer:
The correct answer is 4: "The exception to Mendelian laws of inheritance that best explains the mentioned symptoms is codominance".
Explanation:
In codominance, both alleles can be expressed. In these cases, heterozygote individuals<em> instead</em> of showing an <em>intermediate phenotype</em>, express both of the alleles. Their phenotype is an additive expression of their parents' genes.
In cystic fibrosis, there is a gene responsible for coding for a protein named "cystic fibrosis transmembrane conductance regulator, CFTR".
-Most of the people have two copies of the normal allele and produce the functional CFTR protein form.
-Patients with cystic fibrosis have two copies of the mutated allele and so produce the mutated and dysfunctional form for this protein.
-Heterozygote people possess only one normal CFTR allele and a mutated form for the same allele and produce a normal protein and a mutated protein.
In the last case, both alleles are codominant and they express in heterozygote individuals. Given the fact that the normal allele produces enough functional CFTR protein, these individuals do not have any adverse effect and the mutated allele is recessive at a physiological level.
