Answer:
grow longer
Explanation:
In a long bone, the epiphyseal is the growing region. In young bones, bone formation occurs in a layer of hyaline cartilage. The epiphyseal plate forms cartilage on the epiphyseal end. Cartilage is calcified on the diaphyseal side, and the diaphysis lengthens.
Insect populations can develop resistance to insecticides over time. The evolution of resistance is associated with an increase in the frequency of adaptive genes in the population.
- In the case above described it is expected that a few mosquitoes in the population were resistant to DDT before it was ever used (Option a is correct).
- Dichlorodiphenyltrichloroethane (DDT) is a pesticide used in agriculture.
- After exposure to DDT, those individuals in the mosquito population that didn't carry gene variants (i.e., alleles) associated with the resistance to this pesticide died.
- Subsequently, insects having adaptive alleles associated with DDT resistance survived and reproduced, thereby increasing the frequency of adaptive genes/alleles in the population.
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There are two important regions of a lipid that provide the structure of the lipid bilayer<span>. Each lipid </span>molecule<span> contains a hydrophilic region, also called a polar head region.</span>
I dont know what animation you mean about but i will give my opinion.
ATP are used for energy, they are made inside mitochondria. Mitochondria use oxygen and food to make ATP, which is maybe what u mean by rechargable is because whenever you eat, the mitochondria will produce energy again and again.
Answer:
When a muscle cell contracts, the myosin heads each produce a single power stroke.
Explanation:
In rest, attraction strengths between myosin and actin filaments are inhibited by the tropomyosin. When the muscle fiber membrane depolarizes, the action potential caused by this depolarization enters the t-tubules depolarizing the inner portion of the muscle fiber. This activates calcium channels in the T tubules membrane and releases calcium into the sarcolemma. At this point, <em>tropomyosin is obstructing binding sites for myosin on the thin filament</em>. When calcium binds to the troponin C, the troponin T alters the tropomyosin by moving it and then unblocks the binding sites. Myosin heads bind to the uncovered actin-binding sites forming cross-bridges, and while doing it ATP is transformed into ADP and inorganic phosphate which is liberated. Myofilaments slide impulsed by chemical energy collected in myosin heads, <u>producing a power stroke</u>. The power stroke initiates when the myosin cross-bridge binds to actin. As they slide, ADP molecules are released. A new ATP links to myosin heads and breaks the bindings to the actin filament. Then ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, which starts a new binding cycle to actin. Z-bands are then pulled toward each other, thus shortening the sarcomere and the I-band, and producing muscle fiber contraction.
