Answer:
An explanation inspires new questions and the process of making new observations.
Explanation:
The question is incomplete as it lacks the multiple options. The multiple option are as follows;
Careful monitoring for level of consciousness and resolution of hypoglycemia
.
IV infusion of 50% dextrose and water solution
.
Administration of subcutaneous glucagon
Administration of 15 to 20 g of glucose in a concentrated carbohydrate source
Answer:
Administration of 15 to 20 g of glucose in a concentrated carbohydrate source.
Explanation:
The insulin and glucagon hormone maintains the blood glucose level in the humans. In case of Type I diabetes a little amount or no amount of insulin is made by the pancreas.
The wife of a man has caused insulin error that creates hypoglycemic condition means the individual has low blood glucose level. The intake of carbohydrates can increase his blood glucose level. The wife should give 15 to 20 g of glucose to make the conditions normal.
Thus, the answer is option (4).
Answer:
They difference in phenotypes is dependent on the genes. The more dominant gene will overpower a recessive gene, or if there are two recessive genes together, that will be the phenotype. When things are cross breaded they introduce different genotypes that allow for different looking phenotypes.
Example: Dominant blue flower (Bb) mixed with recessive yellow flower (bb)
B b
b Bb bb
b Bb bb
There is a 50/50 chance the offspring will be either blue or yellow
Whole genes and parts of genes can be extracted from chromosomes, linked to other DNA molecules to form recombinant DNA and introduced into living cells. In a process known as gene cloning, the host cell's biochemical processes are used to make many copies of the inserted gene and the protein it codes for.
<span>Cells control cell division in order to maintain normal cell function. If something happens to the control of the cell division, the healthy cells will divide uncontrollably. These new cells are cancer cells. </span>
The mutations in three genes are responsible for development of cancers:
1. Mutation in proto-oncogenes. Proto-oncogenes normally signal cells to grow and differentiate. Proto-oncogenes can become oncogenes due to mutations which result in the uncontrollable division of the cells.
2. Mutation in tumor suppressor genes. In normal cells, tumor suppressor genes suppress genes essential for cell cycle and that way they prevent uncontrollable cell division. However, after a mutation in these genes, suppression is lost and the cell may progress to cancer.
3. Mutation in stability genes. In normal cells, they have no role either in cell death or growth, but they control mutation rate. Mutation in stability genes results in situation where all genes, including proto-oncogenes and tumor suppression genes, are more frequently mutated.
