Answer:
Explanation:
To do proper research and prepare for it
Answer:
Freckles, no PKU- 9/16
Freckles, PKU- 3/16
No freckles, no PKU- 3/16
No freckles, PKU- 1/16
Explanation:
This question involve two distinct genes; one coding for possession or not of freckles and the other for PKU disease in humans. Allele for freckles (F) and no PKU (P) are dominant over the alleles for no freckles (f) and PKU (p) respectively.
N.B: Possession of freckles is a dominant trait i.e. FF and Ff possess freckles
If a woman who is heterozygous for freckles and a carrier for PKU disease (FfPp) marries a man who is also heterozygous for freckles and a carrier for PKU disease (FfPp), they produce the following gametes: FP, Fp, fP, and fp
Using these gametes in a punnet square (see attached image), the following offsprings will be produced:
Freckles, no PKU (F_P_) - 9/16
Freckles, PKU (F_pp) - 3/16
No freckles, no PKU (ffP_) - 3/16
No freckles, PKU (ffpp) - 1/16
Through suppression of the aconitase enzyme in the tricarboxylic acid cycle, aconitase, fluoroacetate is extremely hazardous to humans and other mammals.
'Lethal synthesis' of an isomer of fluor citrate is the cause. fluoroacetate is present in many different plant species, and ruminant animals can die from ingesting them consists of aconitase.
With the chemical formula FCH 2 CO 2 Na, sodium fluoroacetate is an organofluoride. The taste of this colorless salt, which is utilized as a metabolic toxin, is comparable to that of sodium chloride. Potassium and sodium salts are both derived from fluor acetic acid. In 1942, a report on the efficiency of sodium fluoroacetate as a rodenticide was published.
Learn more about aconitase here
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Umm well carbon dating does how long it was around but, it would be A