Answer:
Genetic mapping for unequivocal identification of the potentially causative mutation
Explanation:
Galactosemia is a genetic disorder caused by mutations in the Galactose-1-phosphate uridylyltransferase (GALT) gene, which encodes an enzyme involved in the metabolism of galactose. Gene mapping is a technique widely used in genetics to identify the position of one locus a chromosome by using molecular markers to estimate genetic distances. Genetic mapping provides useful evidence in order to identify when a disease that is transmitted from parent to offspring can be associated with one or more genes and then determine which gene/s is/are responsible for this condition.
Answer:White blood cells, also called leukocytes or leucocytes, are the cells of the immune system that are involved in protecting the body against both infectious disease and foreign invaders. All white blood cells are produced and derived from multipotent cells in the bone marrow known as hematopoietic stem cells. Leukocytes are found throughout the body, including the blood and lymphatic system.
Explanation:
Answer:
The law of conservation of matter and energy states that matter is neither created nor destroyed but conserved. They can only rearrange the matter and energy. For example, an oxygen atom will cycle through a living system.
The urine osmolality is the measurement of urine absorption,
which substantial values indicate concerted urine and small values specify
weaken urine. Any foods that contained water also affects the osmolality of
urine. Some of the mammals are competent
of higher osmolality than healthy humans, most especially the rats, hamsters,
and mice if given no water.
Several hours after your last meal, declining blood glucose levels stimulate release of the hormone <u>glucagon</u> , which stimulates glycogenolysis, lipolysis and fat mobilization, and gluconeogenesis.
<h3>How does glucagon stimulate gluconeogenesis?</h3>
The biological process through which glycogen degrades into glucose and glucose-1-phosphate is known as glycogenolysis. Hepatocytes and myocytes both participate in the response. Two important enzymes, glycogen phosphorylase and phosphorylase kinase, control the process.
By increasing the activity of hepatic adipose triglyceride lipase, intrahepatic lipolysis, hepatic acetyl-CoA content, and pyruvate carboxylase flux, as well as increasing mitochondrial fat oxidation, glucagon stimulates hepatic gluconeogenesis. All of these actions are mediated by stimulation of the inositol kinase.
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