Answer:
A Recessive
Explanation:
Generally, there are two types of disorders, progressive and recessive. When something is recessive, it is not a dominant gene. The reason why sickle cell anemia is classified as a recessive disorder is because it only affects some people, but it does not affect all those who share DNA. Hope this helps!
<span>If a population is not in Hardy-Weinberg equilibrium that can cause deviations from expectation depending on the assumptions of HW that are violated. If a population violates some of the assumptions (like mutations, migrations and selection) the allele frequencies will change over time. Also, if a non-random mating occurs (like inbreeding), it will cause an increase in homozygosity for all genes.</span>
Answer:
The physical interaction of light with particles and gases in the atmosphere
Explanation:
The way visibilty works is that light bounces off objects and molecules which return to our eyes and give us the ability to see our surrondings and light in the atmosphere is scattered which at times prevents us from being able to see.
Glycolysis requires an investment of 2 ATP, but it yields a total of 4 ATP, netting 2 ATP. So more ATP is produced than is used.
Answer:
a. True, b. False, c.True, d. True
Explanation:
a. Base excision repair is started by a DNA glycosylase that recognizes the changes and removes the altered base by cleavage of the glycosidic bond binding the base and the deoxyribose sugar together.
b. Nucleotide excision repair works by a cut-and patch mechanism that removes their heavy lesions, including pyrimidine dimers and nucleotides . Endonucleases are responsible for the lesion of the damaged strand.
c. Nucleotide excision repair is initiated by the proteins namely UvrA, UvrC, and UvrB in Escherichia coli.
-UvrD (helicase II) later removes the damaged strand
-DNA polymerase I (PolI) fills in the resulting gap.
d. DNA glycolases removes the damaged nitrogenous base.
-It leaves the sugar-phosphate backbone intact and thus creating an apurinic/apyrimidinic site, which is commonly referred to as an AP site.
e. Xeroderma pigmentosum complementation group A(XPA)
-This is an essential protein in the nucleotide excision repair pathway.
- It helps to make a pre-incision complex along with other proteins.
