Answer:
The possible fate of the cell that it may turn cancerous.
Explanation:
The cells present in the body generally work in harmony. However, if a cell attains a mutation, it can make it proliferate in the case when it should not do, and make it thrive in the case when other cells are dying. Due to proliferation, the unusual cell produces more abnormal cells also known as cancerous cells. These cancerous cells become more favorable in comparison to the normal cells due to the phenomenon of natural selection. These cells eventually result in a lethal form of tumors.
In the normal cells, the destructed gene or the damaged cells get repaired easily, in case if the damage is worse the cell dies. A protein known as p53 helps in repairing damaged cells or kills them if the damage is too severe. But in the case of cancer cells, the p53 protein does not work appropriately as they possess a mutated or changed form of p53 protein. Thus, in the case of cancerous cells, the rate of repair lags behind the rate of mutation, which makes the cancer cells thrive and increase in numbers resulting in further destruction.
Answer:
Muscle fatigue is often reported by patients with Marfan syndrome although myopathy is not classically considered a component of Marfan syndrome [1, 2, 4, 6, 7]. In addition to apparent muscle underdevelopment, some patients report myalgia or cramps suggesting skeletal muscle involvement.
5: true
6: Oxygen
7: H+
8: true
Answer:
This is a picture of an aminoacid chain
Explanation:
In an amino acid chain all amino acids ( for example GLY, VAL, or ARG) are linked by peptide bonds. Long chains, also called polymers, of amino acids are called proteins. The enlarged photo is an example of a structure of an amino acid. They have an amine (-NH2) and carboxyl (-COOH) functional group, along with a side chain (R group), specific to each amino acid.
Answer: 133 km/h
Explanation
