Explanation:
Digestion of carbohydrates in the small intestine -
Carbohydrates brake - down from dissachride into the monosaccharide in the small intestine .
The enzyme carbohydratase , carries out these steps where the disacchride are broken into the simpler carbohydrates , that is , the glucose .
This enzyme which is present in the microvilli of the small intestine help in the absorption of glucose have brush border appearance, which increases the surface area of absorption .
Answer:
patient confidentiality, privacy rule, EMR, and health data security
Explanation:
edit:
(Just saw that you figured out lol)
Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’
They grow, divide and die. Cancer cells, on the other hand, don't follow this cycle. Instead of dying, they multiply and continue to reproduce other abnormal cells.