Answer:
The proteins receive electrical signals from nerve cells.
Explanation:
Option A is correct. These ions then have interaction with actin and myosin filaments to purpose muscle contraction. The muscle groups stay in the shriveled state until adenosine triphosphate (ATP) binds to myosin, releasing the myosin and actin filaments from one another.
<h3>How long after a individual dies does rigor mortis set in?</h3>
Rigor mortis appears approximately two hours after dying in the muscle groups of the face, progresses to the limbs over the subsequent few hours, finishing between 6 to eight hours after death. [10] Rigor mortis then stays for some other 12 hours (till 24 hours after death) and then disappears.
<h3>Why does a body go into rigor mortis stiffen within 36 hours of death?</h3>
Rigor mortis is the 1/3 stage in which the muscle mass harden and become stiff, prompted with the aid of the lack of adenosine triphosphate (ATP), which gives electricity to the muscles. Rigor mortis is a extensive tool to any dying examination because it can narrow down the timeframe of death.
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Answer:
The correct statements are that the first calorimeter is reliable but not valid, and the second calorimeter is valid and reliable.
Explanation:
The first calorimeter is reliable as the reading demonstrated by it is similar when each time the experiment is performed, however, the result attained is not correct, though it is reliable. On the other hand, the second calorimeter is both reliable and valid, as it is demonstrating the accurate results from time to time. This is valid as it is providing a similar result as that of the original readings.
Higher trophic level is further up the food chain. Further up you go there are less and less competitors. So the answer is true
Meconium ileus, in newborns, may indicate the presence of cystic fibrosis
Cystic fibrosis is a genetic disorder mainly associated with cells capable of producing mucus. These cells are found in sweat glands, digestive system and lungs belonging to respiratory system. Thus, the abnormality in mucus production, in sticky and thick nature, disrupts the normal functioning of human body. They block the ducts and tubes, which can result in life threatening condition.
The mutation responsible for cystic fibrosis occurs in the gene CFTR, which is abbreviated form of Cystic fibrosis trans membrane conductance regulator. The primary function of gene is to regulate the exchange of fluids and salts present in the cells.
Meconium ileus refers to the inability of bowel movement in the newborn. This occurs due to thick and sticky mucus, which is indicative of cystic fibrosis, as discussed above.
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