B- Electron transport chain
<h2>Problem of protein alters the movement of Ions </h2>
Explanation:
- Proteins are tiny machines that do explicit occupations inside a cell. The directions for building every protein are encoded in DNA. Proteins are gathered from building squares called amino acids.
- The CFTR protein is comprised of 1,480 amino acids. When the CFTR protein chain is made, it is collapsed into a particular 3-D shape. The CFTR protein is formed like a cylinder that experiences the film encompassing the cell, similar to a straw experience the plastic top on a cup.
- In individuals with CF, changes in the CFTR quality can cause the accompanying issues with the CFTR protein:
- It doesn't function admirably
- It isn't delivered in adequate amounts
- It isn't delivered in any way
- At the point when any of these issues happen, the chloride particles are caught inside the cell, and water is never again pulled in to the space outside the cell. When there is less water outside the cells, the bodily fluid in the aviation routes gets got dried out and thickens, making it smooth the cilia. The cilia can't clear appropriately when thick, clingy bodily fluid overloads them.
When looking at a Neuromuscular Junction (NMJ), we know that neurotransmitters (NT) are released from the presynaptic cell and they then bind to the receptors that are located on the postsynaptic cell - this causes the effect of the NT being released.
So we are told that NT are still being released, however they are not having an effect. This would mean that they are probably being blocked by something - in this case, it seems that the neurotoxin is the culprit in the blocking of these receptors.
Therefore, if the NT cannot bind to the receptors on the postsynaptic cell, they are not going to have any effect, no matter how much NT is being released.
So the answer in this case is: The neurotoxin is most likely C) Blocking the receptors on the postsynaptic cell.
B. Gene. Piebalding is caused by mutant copies of genes.
