The results will most likely be valid because the scientists kept getting the same results after REPEATED experiments. If they just conducted one experiment then they would not have been sure if what happened was a fluke or if another thing would happen after another experiment.<span />
Double layered nuclear, the outer nuclear membrane is continuous with the membrane of the rough endoplasmic reticulum (ER), it also continues with the inner nuclear membranes since the two layers are fused together at a numerous tiny holes called nuclear pores that perforate the nuclear enevelope
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The best answer to the question: If every gene has a tissue-specific and signal-dependent transcription pattern, how can such a small number of transcriptional regulatory proteins generate a much larger set of transcriptional patterns? Would be:
Because transcriptional regulators, which are the ones responsible for initiating, and stopping, transcription of RNA into protein, often work in pairs, one goes with the other, and thus increase the regulatory capabilities over gene expression so that the genes translated into RNA and then transcribed into aminoacids in protein chains, actually code for the correct protein types.
These regulators will both stand, as appropriate, on a specific gene to promote its transcription, or prevent it, depending on the different signaling mechanisms received.
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Francium, which has an electronegativity of 0.7
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<em>I think the answer is glucose/sugar</em>
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