Mice that are deficient for Apaf1 or caspase-9 are defective for cytochrome dependent apoptosis. Apoptosis is a critical event in development, allowing excess brain cells to be weeded out. The extent of brain overgrowth and the size of the cranial protrusions indicate that the pruning process in the developing brain must be massive. The dramatic effects of the deficiencies of Apaf1 and caspase-9 suggest that the cytochrome c-dependent apoptotic pathway the intrinsic pathway must be critically important in brain development.
Answer:
Qué secuencia muestra la creciente complejidad de las organizaciones ecológicas?
Explanation:
Option D because Earth is closer to the sun than Mars
Answer:
<em>The correct option is B) Missense mutation.</em>
Explanation:
A missense mutation can be described as a type of point mutation in which a mutation in a single nucleotide causes the codon to change and form a different type of amino acid. As in the above scenario, we can see that the code CAU coded for the amino acid, Histidine, before a point mutation occurred. After the point mutation, the codon was changed to UAU instead of CAU. UAU codes for the enzyme, tyrosine, instead of histidine.
A mutation is a change in trait. For example people who are lactose intolerant are normal, but people that can can drink milk without a problem have mutated. Hopefully this makes sense and helped.
