Answer:
Average atomic mass = 21.38 amu.
Explanation:
Given data:
Mass of first isotope = 21 amu
Abundance of first isotope = 62%
Mass of second isotope = 22 amu
Abundance of second isotope = 38%
Average atomic mass = ?
Solution:
Average atomic mass = (abundance of 1st isotope × its atomic mass) +(abundance of 2nd isotope × its atomic mass) / 100
Average atomic mass = (62×21)+(38×22) /100
Average atomic mass = 1302 +836 / 100
Average atomic mass = 2138 / 100
Average atomic mass = 21.38 amu.
Answer:
A community
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Answer:
Prezygotic barriers:
1. Habitat isolation
2. Behavioral isolation
Explanation:
Pre-zygotic barriers are the barriers which do not allow the formation of the zygote in the organisms.
The two mechanisms of the pre-zygotic barriers are the:
1. Habitat isolation: the Flycatchers do not share the same habitat which is also mentioned in the question that they live on different islands.
2. The behavioural isolation: the Flycatchers could have evolved the different mating rituals as a result of which the could not interbreed.
Thus, Habitat isolation and Behavioral isolation are correct.
<h2>GPCRs and RTKs</h2>
Explanation:
1) Phosphorylation of receptor can terminate signal or desensitize receptor: GPCRs because when signal persists a protein kinase receptor is activated,this further catalyse the phosphorylation of several serine residue at cytosolic phase of 7 transmembrane
Phosphorylated transmembrane recognized by arrestin protein blocks interaction between trimeric G protein and receptor thus blocks signal transduction
2) Autophosphorylation of receptor can initiate signal: RTKs because receptor dimerization activates intrinsic tyrosine kinase activity by which they phosphorylate its own tyrosine residue on cytosolic phase
3) Structure contains seven transmembrane helices: GPCRs because it consists of single polypeptide and spans the membrane 7 times hence called 7 transmembrane;it recognizes external signal and activates trimeric G protein
4) Ligand binding induces conformational change in receptor: RTKs because normally RTK exist in monomeric form in absence of any ligand molecule but when any ligand molecule binds receptor starts to dimerize
5) Receptor activation causes phosphorylation of its cytosolic subunits: RTKs because when receptor gets activates the intrinsic tyrosine kinase activity by which they phosphorylate its cytosolic subunits
6) An example is the insulin receptor: Insulin receptor is present on plasma membrane and belongs to RTK family and always exist in dimeric form
7) Transports some ligands through the membrane: Neither GPCR nor RTK;ligand gets transported through the membrane through ligand gated channel
8) An example is the epinephrine receptor: Epinephrine is an endocrine hormone produced by adrenal glands in stress condition and prepare body for fight and flight;Epinephrine uses GPCR signalling pathway by four different types of membrane receptors in different tissues
9) Activate heterotrimeric G proteins directly: Transmembrane protein of GPCR recognizes the external signal and activates G protein,activated G protein binds with effector enzyme and activate it which further produce or destroy secondary messenger that carry message from cell surface to cell interior
This means that balls with a diamenter of 2.25 inches cannot have any imperfections (bumps or dents) greater than 0.005 inches. In other words, the bump or dent to diameter ratio cannot exceed 0.005/2.25 = 0.0022222
