When a DNA sequence alteration results in a stop codon rather than a codon that specifies an amino acid, it is known as a nonsense mutation. This is rarely seen in 10% of patients with genetic disease.
<h3>What is nonsense mutation?</h3>
A nonsense mutation in a DNA sequence causes a premature stop codon, also known as a nonsense codon, in the transcribed mRNA as well as a shortened, ineffective, and typically nonfunctional protein product.
Because stop codons, also known as nonsense codons, signal the completion of protein synthesis rather than encoding for an amino acid, they are the source of the term "nonsense mutation."
Examples of illnesses for which nonsense mutations have been implicated as contributing factors include: Cystic fibrosis (produced by the G542X mutation in the cystic fibrosis transmembrane conductance regulator); (CFTR) Beta-globin (thalassemia) Hurler disease.
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A list of the cell fibers from largest to smallest would read:
1. microtubules – is a microscopic tubular structure that is present in numbers in the cytoplasm of cells. These are usually aggregated to form complex structures.
<span>2. intermediate filaments – are cytoskeletal components that is usually found in the cells of vertebrate species and </span>other organisms like plants, fungi, unicellular organisms.
3. <span>Microfilaments – are filamentous structures that can be found in the cytoplasm of eukaryotic cells and it will form part of the </span>cytoskeletons<span>. They are composed primarily of polymers of actin. But in cells, they usually interact with other proteins.</span>
38 ATP is produced during cellular respiration
The statement Bonds in the hydrogen peroxide are weakened in catalase's active site, allowing the chemical reaction to occur is the answer. Catalase is an enzyme in the liver that breaks down harmful hydrogen peroxide into oxygen and water.
