When looking at a Neuromuscular Junction (NMJ), we know that neurotransmitters (NT) are released from the presynaptic cell and they then bind to the receptors that are located on the postsynaptic cell - this causes the effect of the NT being released.
So we are told that NT are still being released, however they are not having an effect. This would mean that they are probably being blocked by something - in this case, it seems that the neurotoxin is the culprit in the blocking of these receptors.
Therefore, if the NT cannot bind to the receptors on the postsynaptic cell, they are not going to have any effect, no matter how much NT is being released.
So the answer in this case is: The neurotoxin is most likely C) Blocking the receptors on the postsynaptic cell.
The answer is B because prometaphase is the start of everything hence the "pro" at the beginning of the word
Answer:
Adaptation is process which runs parallel to an evolution
The correct answer is "coarctation of the aorta". In coarctation of the aorta, there is an abnormal narrowing of the aorta, most commonly in the abdominal aorta, which compromises blood flow from going into the lower extremities. Patients with coarctation of the aorta most commonly presents with tachypnea as this is a high pressure congenital heart defect which makes breathing harder for the baby; diminished femoral pulses and poor lower body perfusion as the narrowing compromises blood flow. In physical examination, there will be a narrow ankle-brachial index, or the difference between the pulse pressures of the ankle (lower extremity) and the brachium (upper extremity).
Mitochondria makes the energy for the entire cell, or ATP. Having two or more in a athletes muscle would be a huge advantage because the more ATP, the more endurance the muscle has.
