Answer:
monomers of MONOSACCHARIDES
Explanation:
Polysaccharides are large molecules formed from chains of POLYMERS linked together by glyosidic bonds. <u>MONOMERS are small sub units that formed polymers, they are therefore the building block of a polysaccharides. The monomers of polysaccharides are called monosaccharid</u>es (1 sugar molecule.) when two of these are joined together they formed disaccharides (two sugars.)
Polysaccharides are fromed by joining together condensation, (loss of water molecules,) of mutiple monosaccharides units and the reversal of this to add water molecules to sepate them to monosaccharies is sugar Hydrolysis.
Example of polysaccharides are starch, glycogen cellulose
Example of monosaccharides are glucose, galactose.
Disaccharides are common table sugar, sucrose, maltose, lactose
Explanation:
Organisms don’t have to only reproduce sexually or only reproduce asexually - some animals do both!
When conditions are good, such organisms will reproduce asexually because it is easier. For example, starfish (by fragmentation), slime molds, and water fleas/daphnia (by parthenogenesis) all reproduce asexually when there is plenty of food, minimal predators, and not too much crowding of individuals of the same species.
When conditions worsen (less food, too many individuals, etc), they may switch to sexual reproduction in order to add genetic variation to their population and ensure survival through difficult times.
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Answer:
The gene for human insulin is inserted into the gap in the plasmid. this is done through the use of restriction enzymes and ligase to insert and close the plasmid. This plasmid is now genetically modified. The genetically modified plasmid is introduced into a new bacteria or yeast cell. The more the cells divide, the more insulin is produced as bacteria divides into two identical daughter cells like in mitosis.
Explanation:
Answer:
D. They find more things that are not understood
Explanation:
After discovering something, scientist tend to find something related that sparks their curiousity.
Temperature affects spermatogenesis, which functions best at body temperatures just a little lower than those.
<h3>Abstract:</h3>
To keep testicular temperatures below those of the body core, adequate thermoregulation is essential. The process of mammalian spermatogenesis and the resulting spermatozoa are negatively impacted by elevated testicular temperature. Therefore, sperm quality can be affected and the likelihood of infertility is increased by thermoregulatory dysfunction resulting in heat stress. This article reviews a variety of internal and external factors that may lead to testicular heat stress. We go into more detail on how heat stress affects the spermatogenesis process, the resulting epididymal spermatozoa, germ cells, and the alterations that result in the testis.
We also go over the chemical reactions of germ cells to heat exposure and potential processes, such as apoptosis, DNA damage, and autophagy, that could lead to heat-induced germ cell damage. Further explanation is provided for the intrinsic and extrinsic processes involved in the complex mechanism of germ cell death. These intricate apoptotic pathways ultimately result in the demise of germ cells.
Learn more about spermatogenesis here:
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