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Papessa [141]
3 years ago
11

Which of the following is true of Clydesdale and Thoroughbred horses?

Health
1 answer:
Lorico [155]3 years ago
4 0

 It is C because both Thoroughbreds and Clydesdale horses are in the equine family, but they have different traits. Thoroughbred are the sleek horses you see at horse races. Clydesdales are thinker stocker horses like the ones you see on the Budweiser commercials. I  hope I helped you!

~ Princy

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The recent emergence of molecular pathological epidemiology (MPE), which represents an integration of population and molecular biologic science to gain insights into the etiologies, pathogenesis, evolution, and outcomes of complex multifactorial diseases. Most human diseases, including common cancers (such as breast, lung, prostate, and colorectal cancers, leukemia, and lymphoma) and other chronic diseases (such as diabetes mellitus, cardiovascular diseases, hypertension, autoimmune diseases, psychiatric diseases, and some infectious diseases), are caused by alterations in the genome, epigenome, transcriptome, proteome, metabolome, microbiome, and interactome of all of the above components. In this era of personalized medicine and personalized prevention, we need integrated science (such as MPE) which can decipher diseases at the molecular, genetic, cellular, and population levels simultaneously

MPE is a recently established interdisciplinary and transdisciplinary field. Traditional epidemiology (including molecular epidemiology and genome-wide association studies) has the substantial limitation of treating pathogenically heterogeneous diseases (e.g., hypertension, diabetes mellitus, major depression, breast cancer) as a single entity. In contrast, from the MPE viewpoint, any human disease entity is fundamentally heterogeneous from person to person, just as each individual is unique. Nonetheless, by classifying disease according to its pathogenic mechanisms, we can better predict the course of a disease in a given individual. In fact, there exists heterogeneity of risk factors as well as heterogeneity of molecular pathogenesis in any given disease.

A growing body of literature (see Web Appendix (http://aje.oxfordjournals.org/)) supports this MPE paradigm, with evidence suggesting that carcinogenic or protective effects of lifestyle, dietary, environmental, and genetic factors differ according to specific molecular characteristics in neoplastic cells. The MPE concept is gaining widespread adoption., MPE studies have improved our understanding of pathogenesis by demonstrating consistent links between etiologic factors and molecular subtypes of diseases. Furthermore, recent evidence suggests that host factors can interact with tumor molecular changes to modify cancer cell behavior. Thus, the MPE approach, unlike the traditional epidemiologic research design, allows insights into etiologic factors and pathogenic mechanisms.

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