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Anuta_ua [19.1K]
3 years ago
6

Systole is the ________ phase of the cardiac cycle.

Biology
2 answers:
MrRa [10]3 years ago
8 0
<span>B. Relaxation 

</span>Systole is the ________ phase of the cardiac cycle.
NOT:
A. Contraction
<span>C. Monitoring </span>
<span>D. Termination</span>
arlik [135]3 years ago
3 0

Systole occurs between s1 and s2 and after active filling fill 100% of the ventricles (caused by the sequential contraction created from the SA node). The increased pressure in the ventricles will cause the now lower pressured atrium valves to shut causing s1. Then with all valves closed this creates what is called an Isovolumetric contraction. This occurs when the SA node contraction will travel down the AV node which travels down the septum and eventually reaches the purkinje fibers and causes a simultaneous contraction and open the semilunar valves. This entire process occurs during the systolic phase.


An easy mnemonic to remember is diastolic which is the opposite of systolic sounds like it has the word "die" in it. Dying is when we usually relax and Diastole is the relaxation phase of the cardiac cycle. Systole would therefore be the contraction phase.

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Using the sliding flament theory, explain (or draw) the process of sarcomere shortening. Start from the point where calcium woul
Arte-miy333 [17]

Answer:

  1. Calcium binds to troponin C
  2. Troponin T moves tropomyosin and unblocks the binding sites
  3. Myosin heads join to the actin forming cross-bridges
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  5. The energy is used to impulse myofilaments slide producing a power stroke
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Explanation:  

In rest, the tropomyosin inhibits the attraction strengths between myosin and actin filaments. Contraction initiates when an action potential depolarizes the inner portion of the muscle fiber. Calcium channels activate in the T tubules membrane, releasing <u>calcium into the sarcolemma.</u> At this point, tropomyosin is obstructing binding sites for myosin on the thin filament. When calcium binds to troponin C, troponin T alters the tropomyosin position by moving it and unblocking the binding sites. Myosin heads join to the uncovered actin-binding points forming cross-bridges, and while doing so, ATP turns into ADP and inorganic phosphate, which is released. Myofilaments slide impulsed by chemical energy collected in myosin heads, producing a power stroke. The power stroke initiates when the myosin cross-bridge binds to actin. As they slide, ADP molecules are released. A new ATP links to myosin heads and breaks the bindings to the actin filament.  Then ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, which starts a new binding cycle to actin. Finally, Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.

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