Organism X existed for a short period of time. I really like geology so trust me, I am 100% sure about this question.
(Mark me as the brainiest if this answer helps you!)
Answer: Natural selection is selection due to environmental conditions from random genetic mutations in a population. Among the variants in a population, some will be slightly better adapted to conditions and will eventually out-compete the rest of the population. Survival of the fittest actually means survival of the best adapted, or fittest for purpose, i.e. to survive and thrive.
Explanation: This phrase has caused much miunderstanding of the evolutionary process. ‘Fittest’ has been equated with biggest, or strongest or fastest, but this was a description originating in the nineteenth century, when it was understood quite differently.
Natural selection arises from genetic variation in a population. Jaques Monod, an esteemed scientist, described evolution as where chance and opportunity meet. Chance lies in the genome of a species, random mutations, and the resulting genetic variation in the population.
Opportunity lies in the environment. Environmental change creates opportunities for some, extinction for others. Opportunities can also be from exploiting new environmental niches.
Natural selection is the process by which some random genetic variation gives some of a population a competitive advantage. competition eventually results in one population out-competing all others in its niche. The end-result of competition in nature is monopoly.
But this process continues and further random mutations can result in another population becoming better adapted. That is the actual meaning of survival of the fittest: in modern terms we might say ‘fittest for purpose’, that is, the species orvpopulation best adapted to survive and thrive in that environment.
Answer: DF508 mutation. A Genetic, Hereditary, Autosomal and Recessive Mutation.
Explanation:
Cystic fibrosis (CF) is a recessive autosomal lethal disease, it is most common on Caucasoid populations. Its diagnosis is suggested by the clinical features of chronic obstructive pulmonary disease, persistent pulmonary colonization (particularly with mucoid Pseudomonas strains), meconium ileus, pancreatic insufficiency with or familiarity history of the disease. The FC gene is large, with about 250 Kb of genomic DNA, 27 exons representing about 5% of genomic DNA; encodes a 6.5 kb transcribed mRNA. This mRNA is transcribed into a protein of 1480 amino acid called CFTR (Regulator Transmembrane Conductance Cystic Fibrosis). When a three-base pair deletion, adenosine-thymine-thymine (ATT) identified in the CFTR gene, exon 10, it results in the loss of a single amino acid phenylalanine at position 508 of the protein. This mutation is called DF508; “D” stands for deletion and “F” for phenylalanine amino acid.
