Answer:
T-protease would be active in the stomach and W-protease would be active in the intestines.
Explanation:
Acidic pH's are considered to be less than 7 and 7 is considered neutral. Due to this we know that T-protease is more active in more acidic pH's meaning it would be most active in the acidic stomach.
The answer is She is moving to the traffic light, but she is not moving relative to the bus driver.
Hope I helped
Answer:
65 L
Explanation:
It is assumed that the temperature is constant at both the sea level and the altitude.
<em>According to the gas law, at constant temperature, the pressure of a gas is inversely proportional to its volume.</em> Mathematically:
P1V1 = P2V2 where P1 = pressure at initial volume, V1 = volume at initial pressure, P2 = pressure at final volume, and V2 = volume at final pressure.
In the illustration, V1 = 15 L, P1 = 100 kPa, V2 =?, P2 = 23 kPa
V2 = P1V1/P2 = 100 x 15/23 = 65.22 L
<u>The volume of the hot air balloon at the altitude would be 65 L</u>
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.
