<em>Muscle cells are excitable, meaning they react to stimuli. They may shorten and create a pulling force because they are contractile. Locomotion, facial expressions, posture, and other voluntary motions of the body are all made possible by the contraction of skeletal muscle, which is connected to bones.</em>
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<em>Also, Taylor Swift*</em>
Answer:
No, the child cannot inherit the disease.
Explanation:
The problem tells you that the man has a recessive allele for an inherited disease, but he has a normal phenotype. This means that the disease is recessive and in order for an individual to have the disease, they must have two recessive copies of the allele. The problem also tells you that the mother has a genotype that does not include this allele. With this information, you can do a punnet cross of BB (mother) x Bb (carrier father), and end up with the following possible genotypes: BB, Bb, BB, Bb. Therefore the child will not have the disease, but there is a 50% chance that the child will be a carrier for the disease.
Yes, but some micro organisms that are like that are either too small for scientists to always spot, plus some of them don't just eat the cancer cells. They continue on. You have to realize that decomposes eat and eat on at decaying bodies to the extent that they are just a skeleton. Injecting micro organisms like that into the body is highly dangerous and fatal. A person could slowly die of blood loss if scientists and doctors don't figure out where the organism is and how to get it out of the body. That organism would be in the blood stream making it harder to get out than a tapeworm, that usually hang out in the stomach and taking medication can get them out.
So I would not suggest you tamper with decaying animals. You could get sick without gloves.
Because its is a bit more rare than iron .
<span>Lactase persistence, the ability to digest the milk sugar lactose in adulthood, is highly associated with a T allele situated 13,910 bp upstream from the actual lactase gene in Europeans. The frequency of this allele rose rapidly in Europe after transition from hunter–gatherer to agriculturalist lifestyles and the introduction of milkable domestic species from Anatolia some 8000 years ago. Here we first introduce the archaeological and historic background of early farming life in Europe, then summarize what is known of the physiological and genetic mechanisms of lactase persistence. Finally, we compile the evidence for a co-evolutionary process between dairying culture and lactase persistence. We describe the different hypotheses on how this allele spread over Europe and the main evolutionary forces shaping this process. We also summarize three different computer simulation approaches, which offer a means of developing a coherent and integrated understanding of the process of spread of lactase persistence and dairying.</span>