Answer:
B
Explanation:
The Krebs cycle fuels the production of ATP, aka energy. Hope I helped :)
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
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The sodium amytal test involves the injection of a small amount of sodium amytal into the carotid artery on one side of the neck. This injection anesthetizes the hemisphere on that side for a few minutes.
A method that involves injecting a small amount of a barbiturate into the carotid artery on one side of the head to assess hemispheric functions, usually memory and language. The cerebral hemisphere that was injected selectively becomes impaired for 10 to 15 minutes during this operation.
Various cognitive tests are given while each hemisphere is seperately anaesthetized; deficiencies on these tasks imply that these functions are represented in the anaesthetized hemisphere. Prior to a temporal lobectomy, the Wada test may be utilised in cases with severe and uncontrollable epilepsy. Also known as the Wada technique, intracarotid sodium Amytal test (ISA), Wada dominance test, and intracarotid amobarbital procedure.
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The answer should be <span>For gripping materials</span>
