Photosynthesis takes in sunlight and carbon dioxide, along with water and nutrients, so the plant can make energy. That energy is used for cell growth and repair, producing seeds, etc. The plant will exhale oxygen as a byproduct of photosynthesis.
Similarly, cellular respiration is also used to create energy for the host organism. However, the cells take in oxygen along with something like complex sugars (ie the stuff you eat) and use those components to form energy for the cell. The energy for human cells is useful for cell growth and repair, nerve impulses, muscle contractions, etc. Basically anything you can think of that the human body needs to do on a regular basis, it requires energy to do so. A byproduct of cellular respiration is carbon dioxide, which is exhaled out to the environment. The plant will then absorb the CO2 to keep the cycle going.
In short, both processes are used to make energy for the cell. The differences is what the inputs for each process require. Photosynthesis takes in sunlight, water, and carbon dioxide (and other plant nutrients), and exhales oxygen. Cellular respiration takes in oxygen and complex sugars (from plant material or other animals) and exhales carbon dioxide.
Answer:
C: They all have some mutation in some wing-development gene. Each strain may have a different mutation in a different gene, but all strains within a complementation group have the same phenotype.
Explanation:
From the situation of the question given, we can say that the true statement about flies that belong to different complementation groups is option C primarily because Strains existing in different complementation groups obviously would have mutations in different genes.
Answer:
BRCA1 and BRCA2 genes
Explanation:
The BRCA1 and BRCA2 genes are tumor suppressor genes that encode breast cancer susceptibility proteins involved in repairing double-strand DNA breaks (DSBs) by the mechanism of homologous recombination. This mechanism uses the sister chromatid of the homologous chromosome as template for repairing DSBs. It has been shown that mutations in BRCA1 and BRCA2 genes alter the molecular mechanism of homologous recombination DNA repair, thereby mutations are not repaired appropriately and persist in the DNA of tumor cells.
A
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