Answer: A virus lack the metabolic machinery common to other microorganisms
Explanation:
A virus is an ultramicroscopic particle capable of causing disease. It differs from other disease-causing organisms like bacteria, nematodes etc, because it has no metabolic system of its own but relies on its host system for survival and replication.
Hence, it is difficult for antibiotics to target and destroy viruses without harming patients, rather antiviral drug are prescribed against viruses
B) Prairie grasses have narrow leaves.
Took the quiz it's right.
Answer:
A. A majority of scientists agreed with it
Explanation:
The famous theory called the theory of common descent, states that all the living organisms of the earth have arisen from a common ancestor. This notion was first proposed by a French mathematician,Louis Maupertuis duirng 1740s who was of the view that all organisms had a single ancestor and evolutionary process with the passage of time resulted in the specie diversification.
After that, in 1790s another philosopher Immanuel Kant, suggested that all organisms seem to have a common ancestor. In the same period of 1790s, another scientist , Erasmus Darwin who was the grandfather of Charles Darwin also suggested that all the organisms might have a single ancestor who went through the process of evolution to bring all the majesty into life.
Charles Darwin was the first scientist who worked on this notion for alot of time and proposed the theory of common descent,in his book, On the Origin of Species.
After it, many scientists got agree with this theory such as, Vernon Kellogg in 1907 and T. Ryan Gregory in 2008 and many others explain that no reliable observations exists which contradicts the theory of common descent.
Therefore, option A is the best option.
Hope it helps!
Answer:
down syndrome
Explanation:
people with down syndrome have an extra chromosome in the twenty-first pair
Protein-protein interactions within the CARMA1-BCL10-MALT1 complex:
- The T-cell receptor and B-cell receptor-dependent NF-B induction and lymphocyte activation are mediated by the CBM complex, which is made up of the proteins CARMA1, BCL10, and MALT1.
- Each of the proto-oncoproteins CARMA1, BCL10, and MALT1 is a somatic gain-of-function mutation or chromosomal translocation, and dysregulation of CBM signaling is a characteristic of numerous lymphoid malignancies, including Activated B-cell Diffuse Large B-cell Lymphoma.
- Moreover, a number of immunological dysregulation diseases have been linked to both gain- and loss-of-function germline mutations in CBM complex proteins.
- Over the past ten years, careful examination of the interactions of CBM components has yielded a wealth of detailed structural knowledge.
- Here, we discuss important discoveries about the molecular nature of these protein-protein interactions that have helped the research develop a detailed understanding of how these proteins come together to form high-order filamentous CBM complexes.
- Approaches to therapeutic suppression of the CBM complex have thus far centered on obstructing MALT1 protease activity in order to treat lymphoid malignancy and/or autoimmunity.
- The structural effects of MALT1 protease inhibitors on significant protein-protein interactions are also reviewed in detail.
To learn more about protein-protein interaction visit:
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