Given what we know, we can confirm that ready-to-eat TCS food must be date marked if it will be stored for longer than 24 hours.
<h3>What is TCS food?</h3>
- TCS food refers to food that must be stored in temperature-controlled environments to ensure safety.
- These foods, if not stored properly and at the correct temperatures can cause sickness to the consumers.
<h3>Why does it need to be date-marked?</h3>
- The reason the foods need to be date-marked is to relay information to the potential consumer as to by when the food should be consumed.
- This is done to ensure that if stored correctly, the food does not have time to spoil and cause health risks for consumers.
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Therefore, we can confirm that ready-to-eat TCS food must be date marked if it will be stored for longer than 24 hours in order to <em><u>protect the </u></em><em><u>health </u></em><em><u>of the </u></em><em><u>consumers</u></em><em><u>. </u></em>
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To learn more about TCS food visit:
brainly.com/question/4247127?referrer=searchResults
The characteristics of T lymphocytes that are involved in specific immunity includes;
They mature in the thymus
They move freely among lymphoid tissues and connective tissues
They are responsible for cell mediated immunity.
Specific immunity is a subsystem of immune system that is comprised of highly specialized, systemic cells and processes that prevent the growth of pathogens or eliminate them from the body.
I believe its D. response neuron.
Hope this helps!
Love, Grace-
<span>Lafora disease is the most severe teenage-onset progressive epilepsy, a unique form of glycogenosis with perikaryal accumulation of an abnormal form of glycogen, and a neurodegenerative disorder exhibiting an unusual generalized organellar disintegration. The disease is caused by mutations of the EPM2A gene, which encodes two isoforms of the laforin protein tyrosine phosphatase, having alternate carboxyl termini, one localized in the cytoplasm (endoplasmic reticulum) and the other in the nucleus. To date, all documented disease mutations, including the knockout mouse model deletion, have been in the segment of the protein common to both isoforms. It is therefore not known whether dysfunction of the cytoplasmic, nuclear, or both isoforms leads to the disease. In the present work, we identify six novel mutations, one of which, c.950insT (Q319fs), is the first mutation specific to the cytoplasmic laforin isoform, implicating this isoform in disease pathogenesis. To confirm this mutation's deleterious effect on laforin, we studied the resultant protein's subcellular localization and function and show a drastic reduction in its phosphatase activity, despite maintenance of its location at the endoplasmic reticulum.
I got my information from </span>https://www.ncbi.nlm.nih.gov/pubmed/14722920
