Answer:
A. play a role establishing and maintaining membrane lipid asymmetry
Explanation:
Proteins that move move certain phospholipids between leaflets are transmembrane proteins via "flip-flop" transition have also the possibility to maintain membrane lipid asymmetry. There are few types of such proteins:
- flipases-move lipids from the exterior to the interior side of the membrane, energy required
- flopases-move lipids in reverse comparing to flipases, energy required
- scramblases-no energy required, bidirectional
Because of the action of those proteins, cytosolic side of plasma membrane differs from exoplasmic side (asymmetry).
Answer:
Explanation:
Abstract. The use of antibiotics in food animals selects for bacteria resistant to antibiotics used in humans, and these might spread via the food to humans and cause human infection, hence the banning of growth-promoters.
Bowling bc ur not reacting to something thats directly coming at you
for an example catching the ball in basketball and having to shoot it right after
This question is incomplete. The full question is:
Trinucleotide repeat disorders are hereditary diseases caused by mutant genes containing an increased number of repeats of a DNA trinucleotide sequence. Which sequence(s) contain a trinucleotide repeat
a)...CAC GGA AGA AGA AGA AGA AAT AGAC...
b)...AGC GAC AGC AGC AGC AGC AGC AAG T...
c)...TTC ACT GTC ACT GTC ACT GTC ACT GTC C...
d)...CAC GGC GGC GGC GGC GGC ATC GC...
e)...GGC AGGC AGG CAG GCA GGC AGG CTG...
Answer:
a)...CAC GGA AGA AGA AGA AGA AAT AGAC...
b)...AGC GAC AGC AGC AGC AGC AGC AAG T...
d)...CAC GGC GGC GGC GGC GGC ATC GC...
Explanation:
Trinucleotide repeat is a genetic mutation where a crack of three nucleotides is repeated, next to each other, so many times that it leaves the DNA molecule unstable.
This mutation is capable of causing errors in the reading of DNA, which results in disorders and syndromes in the affected organism.
The three strands of DNA shown above show this mutation.
Thickening of gill epithelia in rainbow trout, caused by chloride cell proliferation, could lead to an impairment of oxygen uptake under moderate to severe hypoxia (Thomas et al 1988; Bindon et al., 1994; Greco et al., 1995).
<h3>What results in an increase in AMS in interstitial lung disease?</h3>
The number of alveolar macrophages (AMs) can rise in interstitial lung disease. Precursor cells from the peripheral circulation may have been drawn in, and/or there may have been local lung growth, to create this.
<h3>What connection does sarcoidosis have between lymphocytes and proliferating cells?</h3>
Additionally, a strong association between the quantities of lymphocytes and proliferative cells in sarcoidosis and fibrosis was discovered in bronchoalveolar lavage (BAL). Eosinophil counts and proliferating cell counts were positively associated in fibrosis.
<h3>How do AMS patients and healthy controls differ in terms of propagating AMS?</h3>
With a substantial association between these two indices, there was a considerable increase in proliferating AMs in all patient groups when compared to healthy controls (4.2 versus 1.4% Feulgen, and 2.1 against 0.5% Ki67).
Learn more about cell proliferation:
brainly.com/question/19551481
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