Answer:
The reference point of happiness is derived from personal perceptions.
Explanation:
Perception is a product of the individual's learning, memory, expectation, and attention to sensory signals. Individual perceptions are shaped by many factors. One factor relates to the fact that individuals have some differences in how they assess their emotional states. Some individuals focus on themselves, while others focus on other people. The loci of focus affect how they perceive their happiness. Another factor is the way individuals process information. Some process information quickly while other people take time to think and process the information about their states of happiness. The depth of attribution also differs from one person to another. This suggests that while somebody may be considered to be unhappy, the same individual may be enjoying his or her life without qualms.
Answer:
There are 20 intial teeth in child’s mouth.
Sinus tachycardia can be the most probable condition which results into high heart rate above 105 beats/minute. This problem is not very serious and is treatable.
<h3>What is Sinus tachycardia?</h3>
Sinus tachycardia is a type of irregular heartbeat which is characterized by a faster than the normal heart rhythm. The heart's sinus node generates electrical impulses which travels through the heart muscle that causes the heart to beat. A normal sinus rhythm has an average heart rate of the range between 60 and 100 beats/minute.
Treatment is not necessary for sinus tachycardia as it is not a very serious issue. However, if an underlying condition is causing these symptoms, it needs to be treated on time. Treatments for sinus tachycardia include medications such as beta-blockers or calcium channel blockers which can lower the heart rate.
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Answer:
Edema occurs when an excessive volume of fluid accumulates in the tissues, either within cells (cellular edema) or within the collagen-mucopolysaccharide matrix distributed in the interstitial spaces (interstitial edema)
Explanation:
The definition of edema is a swelling due to the expansion of interstitial fluid volume in tissues or an organ. Several clinical conditions present with edema, making it a critical clinical feature for diagnostic medicine. Edema can present in numerous forms including unilateral, bilateral, localized, or generalized edema. Therefore, it is vital to assess the unique presentation and mechanism of edema to understand how it relates to disease pathophysiology, clinical presentation, and treatment. This review will present an overview of the general and cellular characteristics of edema, the mechanism, and pathophysiology of edema, and how edema relates to a specific disease presentation and development.
Answer:
Bacteria are highly adaptable microorganisms who have the capability of developing defense mechanisms against that which may harm them. Not least important of all, is the easiness with which some bacteria, especially pathogenic bacteria like Salmonella, or Klebsiella, develop mechanisms of resistance to antiseptics and, most importantly, antibiotics.
Antibiotics are a chemical substance that was created, and has been developed, in order to be able to combat pathogenic microorganisms, specifically bacteria. However, because today these substances are being used indiscriminately, we are now seeing a very worrying pattern of antibiotic-resistance patterns in microorganisms that used to be sensible to them. The result, we are facing strains of pathogenic bacteria, like Klebsiella pneumonia and E. Coli, that have become resistan to all types of antibiotics, from first generation, to fourth generation. And this has meant that when people acquire infection by these pathogens, the likelihood of death by them has increased because there are no agents capable of combating them.
Exposure to antibiotics has been the sole reason why these resistant strains of bacteria have emerged, especially when these antibiotics are not necessary. And feeding these substances to animals, to ensure their development and weight gain, has not made the situation any better. Now, we are instead adding also bacteria to the list that did not use to be resistant, but that are becoming so as they become adjusted to the constant exposure to antibiotics. Again, the result has been: more people infected with bacterial strains that cannot be combated with any of the existing antibiotic agents.