1. Pulmonary Blastomycosis: is transmitted from inhalation of spores from dust and iscaused by Blastomyces dermatitidis. It targets the lungs and shows signs of fever,chills, joint pain, muscle pain and headache and cough.
2. Histoplasmosis:caused by Histoplasma Capsulatum that is transmitted by theinhalation of spores near bird or bat droppings, it targets the alveolar in the lungs andshows signs related to the flu, and chronic cough.
3. Aspergillosis: this is caused by the organism aspergillus spp. It is transmitted by theinhalation of spores from moldy vegetation and targets the lungs. The symptoms arecough with blood, wheezing, fatigue, shortness of breath, fever and chills, chest pain,face swelling and skin lesions.
Answer:
Explanation:
It maintains the integrity of the cardiomyocyte as it turns a permeability barrier that provides a physical interface between the inside and outside of the myocte
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
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Proteins are the main structural and functional components of cells.
Each group of three bases in mRNA constitutes a codon, and each codon specifies a particular amino acid. The mRNA sequence is thus used as a template to assemble in order the chain of amino acids that form a protein.
The sequence of a protein is determined by the DNA of the gene that encodes the protein change in the gene's DNA sequence may lead to a change in the amino acid sequence of the protein. The sequence of amino acids of a protein determines protein shape, since the chemical properties of each amino acid are forces that give rise to intermolecular interactions to begin to create secondary structures, such as α-helices and β-strands.
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