Answer:
B) a nonsense mutation; this is because a nonsense mutation results in the change of a regular amino acid codon into a stop codon, which ceases translation. This fits with the problem's description of the protein that causes the symptoms as too short, as translation is the process by which proteins/polypeptides are created. A missense mutation would not be the answer because it still codes for an amino acid, which would not shorten the protein. A duplication of the gene would probably just lengthen the protein or not affect its length at all.
Answer:
1. transcription of Tau DNA in different directions.
4. different chemical modifications of Tau protein.
Explanation:
Tau gene transcribe in different direction which depends upon the animal age. This happens because at different age and developmental stage, animal produce different RNA binding proteins which leads to formation of alternative splice forms. This reaction is strengthen by the addition of phosphate and sugar molecules which again depending on developmental stage and time.
I found the answer to be cells
Answer: Characteristics: The fins are supported by rays, as the name indicates. In contrast to the cartilaginous fish they have a rigid skeleton. The swim bladder is also a unique feature of most ray-finned fish, enabling them to maintain buoyancy as they move up or down in the water.
