Prader-Willi syndrome (PWS) is a gentic disorder which has an impact on numerous physiological systems. PWS affected individuals (specifically babies) experience delayed growth, significant hypotonia (low muscle tone), and feeding issues. It does effect circadian rhythms in mice models.
SNORD116, often referred to as HBII-85, is a non-coding RNA (ncRNA) molecule that contributes to the alteration of other small nuclear RNAs (snRNAs). Unlike the majority of other snoRNAs, SNORD116 is not significantly complementary to ribosomal RNA and is expressed widely in the brain (but not in PWS patients).
According to the studies, SNORD116 cause sleep defect in patients with Prader-Willi syndrome. Same observation was seen in mouse models too. Paternal expression of SNORD116 is thought to be a potential gene for the sleep disruptions/circadian rhythm’s that the majority of PWS sufferers.
To learn more about circadian rhythm click here
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My guess is D. It’s between that and C. Put D though
Answer:
Option (a) and (d).
Explanation:
The breaks in DNA molecule may occur due to replication error and oxidizing agent. The double-stranded breaks in DNA can be corrected by Homologous end joining and non homologous end joining.
Homologous end joining is used to repair the DNA present in G2 nad S phases of the cell cycle. The homologous sequences of DNA is used to repair the DNA. Non homologous end joining occurs in the cell present at G0 and G1 phase of the cell cycle. The DNA broken ends are juxtaposed and later rejoin together by DNA ligase.
Thus, the correct answer is option (a) and (d).
They originate near the Equator!
Answer:
humanity has a 95% probability of being extinct in 7,800,000 years
