When looking at a Neuromuscular Junction (NMJ), we know that neurotransmitters (NT) are released from the presynaptic cell and they then bind to the receptors that are located on the postsynaptic cell - this causes the effect of the NT being released.
So we are told that NT are still being released, however they are not having an effect. This would mean that they are probably being blocked by something - in this case, it seems that the neurotoxin is the culprit in the blocking of these receptors.
Therefore, if the NT cannot bind to the receptors on the postsynaptic cell, they are not going to have any effect, no matter how much NT is being released.
So the answer in this case is: The neurotoxin is most likely C) Blocking the receptors on the postsynaptic cell.
Are you sure these are the only answer choices? I believe the answer would be Hippocampus, but it's not up there.
epididymis-vas deferens-urethra
The correct answer is "i<span>t receives incoming sound information and directly stimulates the auditory nerve to transmit information to the brain."
Cochlear implants function to bypass the damaged cochlea due to sensorineural hearing loss (i.e. chronic noise exposure). Cochlear implants functions just like the hair cells and capture sound and transmit it via the vestibulocochlear nerve or the cranial nerve VIII and then to the temporal lobe in the auditory cortex.
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Answer:
Biogenesis
Explanation:
The theory of biogenesis states that only living organisms can produce other living organisms.
