Answer:
sharks are not cold blood killers because they don't kill for fun they kill because they are confused because when they see a human in the water swimming or splashing so they might try to investigate but sharks have more to fear to us than we do to them
::) hope this helps
Answer:
True
Explanation:
Hyperglycemia which means high Glucose level in the blood as a result of lack of insulin can damage the glomeruli. This interfere with the function of the glomeruli. Normally glomeruli fiters blood and remove nutrients to form urine but hyperglycemia will affect it's negatively and will make protein to leak from the blood into the urine. This can also lead to glomeruli called glomerulosclerosis.
Yeast lacks a membrane-bound nucleus. The onion skin cell has one
Hello, I figured your question was missing its options so I went online to find them. Here they are:
The process of phagocytosis involves all of the following EXCEPT
:
a. adhesion.
b. secretion of cytotoxins.
c. elimination.
d. vesicle fusion.
e. chemotaxis.
Answer:
The correct answer is: b) secretion of cytotoxins.
Explanation:
Phagocytosis is a mechanism performed by cells in which the plasma membrane engulfs a large particle. Phagocytosis is used by cells in the immune system to ingest pathogens like viruses and bacteria.
Phagocytosis consists of many steps:
- activation
- the phagocytes that were resting are activated in the inflammatory response when a pathogen enters the body.
- chemotaxis - this refers to the process in which the phagocyte moves to the pathogen by following the chemical factors released by these germs.
- adhesion - the phagocyte attaches to the pathogen.
- ingestion
/vesicle fusion - the phagocyte sends pseudopods to engulf the pathogen, and places it in a phagosome, which is an endocytic vesicle. The phagosome and the phagocyte will fuse so the pathogen gets inside.
- elimination - the pathogen is destroyed in the phagocyte by the lysosomes present in it.
<u>The</u><u> secretion of cytotoxins</u><u> is not a part of the phagocytosis, and is a process exclusive to </u><u>T cells</u><u> (leukocytes that lack the ability to phagocyte).</u>
Answer:
1. Acetylcholine binds to receptors on the motor end plate
2. Ligand-gated channels open leading to depolarization
3. End plate potential triggers an action potential
4. Transverse tubules convey action potentials into the interior of the muscle fiber
5. Calcium is released from the sarcoplasmic reticulum
6. Calcium ions bind to troponin, which then moves tropomyosin
Explanation:
Acetylcholine (ACh) is a signaling molecule (neurotransmitter) that binds to receptors on muscle cells. This binding triggers the opening of ligand-gated sodium channels, thereby ions enter into muscle cells, which causes the depolarization of the sarcolemma and thus promotes the release of Ca2+ ions from the sarcoplasmic reticulum. The myoneural junction, also known as the motor endplate, is the site of synaptic contact between a motor axon and a skeletal muscle fiber. The endplate potential is the voltage that produces the depolarization of muscle fibers when ACh molecules bind to their receptors in the cell membrane. This depolarization spreads in the sarcolemma through transverse tubules (T tubules) and thus generates an action potential. Finally, this action potential induces the release of Ca2+ in the sarcoplasmic reticulum, which activates troponin protein and induces muscle contraction.
