Answer:
Some diseases are more common in certain groups of people, such as Caucasians or African Americans because individuals in such ethnic groups often share certain alleles (versions of their genes), that have been passed down to them from common ancestors and a particular genetic disorder may be more frequently seen in such groups if one of these shared genes contains a disease-causing mutation.
Explanation:
Some genetic diseases are frequently seen in certain ethnic groups like Caucasians or African Americans. Individuals in such groups often share certain alleles (versions of their genes), that have been passed down to them from common ancestors and one of these shared genes may contains a disease-causing mutation.
Examples of certain genetic disorders that are more common in particular ethnic groups include the Tay-Sachs disease, which is more common in people of eastern and central Europe (Ashkenazi), Jewish or French Canadian ancestry and the sickle cell disease, which occur among people of African, African American, or Mediterranean heritage.
Some genetic disorders are more common in people whose ancestry can be traced to a particular geographic area. The factors that can lead to development of populations with very different genetic allele frequencies include their geographic origin, selection, patterns of migration, historic events, etc. Certain natural barriers like oceans and other water bodies, high mountains, large deserts, or major cultural factors had prevented communication and interaction between people. So mating was restricted within the group, and this produces genetic marker differences and differences in the presence of specific disease-related alleles.
A macrophage may engulf a pathogen by endocytosis, which involves the reorganization of actin microfilaments in the membrane and microtubules in the cytoskeleton.
<h3>What is endocytosis?</h3>
Endocytosis is a cellular process by which a cell can absorb materials from the surrounding medium, such as viruses and bacteria in the case of macrophages.
Microfilaments and microtubules are proteins that form part of the cytoskeleton and dynamic conformational changes of them are associated with the cytoskeletal rearregement during the phagocytic process.
The actin microfilaments in the membrane and microtubules in the cytoskeleton form part of the process of endocytosis because they need to reorganize during cell movement.
In conclusion, a macrophage may engulf a pathogen by endocytosis, which involves the reorganization of actin microfilaments in the membrane and microtubules in the cytoskeleton.
Learn more about actin filaments here:
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Sickle cell disorder is an inherited blood disease featured by defective hemoglobin. The condition affects the hemoglobin, or the red blood cells, and their tendency to conduct oxygen. The normal hemoglobin cells are round, smooth, and flexible, and thus, they can travel via the vessels in the body effortlessly.
On the other hand, the sickle cell hemoglobin cells are sticky and stiff and form a sickle, C shape, when they lose their oxygen. These cells combine together, and cannot effortlessly move through the blood vessels.
Answer:
the answer is A. E. coli B
Explanation:
The multiplicity of infection (MOI) refers to the ratio between the numbers of viruses used to infect <em>E. coli</em> cells and the numbers of these <em>E. coli </em>cells. Benzer carried out several experiments in order to define the gene in regard to function. Benzer observed that <em>E. coli </em>strains with point mutations could be classified into two (2) complementary classes regarding coinfection using the restrictive strain as the host. With regard to his experiments, Benzer observed that rII1 and rII2 mutants (rapid lysis mutants) are complementary when they produce progeny after coinfect E. coli K (where neither mutant can lyse the host by itself). The rII group of mutants studied by Benzer does not produce plaques on <em>E. coli</em> K strains that carry phage λ (lysogenic for λ), but they produce plaques on <em>E. coli</em> B strains. This study showed that rIIA and rIIB are different genes and/or cistrons in the rII region.
