I believe the answer is B: <span>Homologous chromosomes are separated from each other to form diploid daughter cells. But, correct me if I'm wrong.</span>
Answer:
Oxygen and glucose are both reactants in the <u>Process</u> of cellular respiration.
ATP is the main Product formed in cellular respiration, and waste products include carbon dioxide and water.
Hope this helps!!
The <span>ability of leukocytes to move in and out of blood vessels in order to reach sites of inflammation or tissue destruction is known as leukocyte extravasation (also called <em>diapedesis</em><u />).</span>
Answer:
False
Explanation:
The cerebral cortex refers to the outer layer of the brain. This layer is formed by a thin film of nervous tissue that surrounds the surface of the cerebral hemispheres, with primates having a much more developed cerebral cortex than other animals.
the cerebral cortex is a thin layer of neurons and neuronal connections which is embedded in the neocortex(the neuronal cells of the cerebral cortex which form in the deep layer), it is not homogeneous, since it is made up of six layers of cells, each with specific and specific functions.
Answer:
1. Inhibiting IP3 channels, leading to decreased Ca2 in the sarcoplasm and reduced contraction.
2. Increasing the relative activity of MLCP, leading to a decrease in tension.
3. Activating K channels, increasing K leaking out of the cell which hyperpolarizes it and decreases the likelihood of Ca2 entry.
Explanation
In smooth muscle, cyclic AMP (cAMP) mediates relaxation because cAMP inhibits a specific kinase required for myosin light chain protein (MLCP) phosphorylation, thereby triggering contraction in the smooth muscles. It has been shown that cAMP inhibits 1,4,5-trisphosphate (IP3)-dependent calcium ions (Ca 2+) release by activation of the cGMP-dependent protein kinase (PKG). PKG proteins act to modulate Ca2+ oscillations by stimulating sarcoplasmic Ca2+-ATPase membrane proteins, increasing Ca2+ in the sarcoplasmic reticulum stores and Ca2+ efflux from the cells, and activate voltage-gated potassium (K) channels, thereby leading to membrane hyperpolarization and reducing Ca2+ entry through Ca2+ channels.
