Individuals with glucose-6-phosphate dehydrogenase deficiency have increased resistance to malaria because the parasite does not survive well in cells with oxidative stress.
<h3>What is the link between G6P deficiency and malaria?</h3>
- Glucose-6-phosphate dehydrogenase (G6PD) deficiency is an X-linked recessive disorder.
- G6PD protects cells from oxidative damage especially the RBCs whose main function is to carry oxygen.
- Mutation in the gene results in G6PD deficiency leading to oxidative stress which can lead to anemia (iron deficiency) as there is breakdown of RBCs.
- However, G6PD deficiency is quite common in parts of Africa, Middle East, and South Asia, which are the regions of high malaria endemism.
- This is due to natural selection in which G6PD deficiency is not eliminated from the population as it can protect people from malaria infection.
- Malaria parasite <em>Plasmodium</em> species does not survive well in cells with oxidative stress.
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Answer:
In strict mode, the narrow-sense heritability is the proportion of the additive genetic variance that contributes to the total of the phenotypic variance. This value can be associated with the inheritance of the a-thalassemia
.
Explanation:
A-thalassemia is a genetic disorder caused by mutations affecting four different genes that encode alpha-globin, thus affecting the hemoglobin production process and, consequently, oxygen transport. The mode of inheritance of the a-thalassemia may be associated with narrow sense heritability since the phenotype is manifested by gradation, i.e., each allele might contribute in similar mode to this genetic condition.
Anaerobic breakdown of glucose produces lactic acid, which accumulates in the tissues and blood.
