The true statements are:
1) Catabolism is the process in which complex substances are broken down, yielding a net output of energy.
Catabolism reactions break down macromolecules (lipids, proteins, nucleic acids and sugar) into smaller units that can be used for further reactions, for energy, for the synthesis of new polymer molecules or be simple waste products.
2) Metabolism is an integrated system in which many of the same reactions participate in degradative (catabolic) and biosynthetic (anabolic) pathways.
Metabolism combine all chemical reactions of an organism. Functions of metabolism include: providing energy form food molecules, synthesis of building blocks of the cell, elimination of the waste products.
3) Anabolism involves the synthesis of polymeric biomolecules and complex lipids from monomers such as fatty acids and nucleotides.
Anabolic reactions are opposite of those in catabolism: these reactions require energy (endergonic processes) for the synthesis of necessary molecules for the cells. That energy is the product of catabolic reactions.
Answer:
Two locus
Explanation:
Let assume the gene for white (1st locus) be W i.e. ww = recessive
here, Let the alleles for the 2nd locus be B and b.
White: W_B_, W_bb
Black&White: wwB_
Black: wwbb
This is dominant epistasis. In dominant epistasis, where the dominant allele of the 1st locus (W) masks or hide the expression of the 2nd locus. When the two alleles in the 1st locus are recessive (ww), the alleles in the 2nd locus can be expressed(B and b).
The most useful microscope to use would be a Compound Light Microscope. These microscopes are limited to about a good 2000X magnification. Not only can you use this to estimate the number of red blood cells, but it is good for seeing and identifying protozoa, bacteria, and organism cells.
Answer:
Hypermethylation of___ promoters will lead to reduced expression of the correspondent genes. Some genes such as Cell Cycle stop genes _and__DNA repair genes_need to be expressed. When they__are not expressed_, this can lead to cancer.
Explanation:
Oncogenes are genes that may trigger cancer, where they are generally overexpressed (i.e., expressed at high levels). Although there are well-documented exceptions to this rule, DNA methylation generally represses gene expression by preventing the binding of transcription factors to promoters. It is known that the hypermethylation of tumor suppressor genes including, for example, cell cycle stop genes and DNA repair genes, may lead to cancer. Similarly, hypomethylation of oncogenes such as, for example, Cyclin D1 (a cyclin-dependent kinase involved in cell cycle progression), may also contribute to carcinogenesis.
