The sarcomere will have uncontrolled muscle contractions.
Troponin blocks myosin-binding sites in the tropomyosin of the actin. If the troponin is damages, the myosin-binding sites will be exposed and myosin heads (of the myosin fiber) will be able to form cross-bridges with the actin even when not required.
Explanation:
This means the sarcomere will contract even when not required or expected hence resulting in symptoms like muscle spasms. Usually, troponin molecules exposes the myosin-binding sites on the tropomyosin upon binding of Ca²⁺ ions. These Ca²⁺ are usually released by the sarcoplasmic reticulum upon impulse from the inverted nerve.
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<h3><u>Answer;</u></h3>
Preload, afterload, and contractility
<h3><u>Explanation;</u></h3>
- Cardiac output is the amount of blood the heart pumps in 1 minute, and it is dependent on the heart rate, contractility, preload, and afterload.
- Preload is the initial stretching of the cardiac myocytes or muscle cells prior to contraction. Afterload is the force or load against which the heart has to contract to eject the blood.
- Contractility is the intrinsic strength of the cardiac muscle independent of preload, but a change in preload will affect the force of contraction.
There is a much greater surface area for the exchange of oxygen and carbon dioxide.
The answer is bacterial cell
The producer would be
A) flower