Answer:
I believe that they are becoming more concerned for zoophytic disease because it is becoming more and more common for these animal transmitted disease to spread, such as ringworm or hookworms. The factors that would make these more of a threat would likely be the amount of people with animals or the other animals or people their animals are around or not taking your animal to the vet upon signs of seeing symptoms of declining health
Explanation:
Tympanometry, a cochlear implant, Toni merry
The consequences if presynaptic action potentials In an axon release insufficient acetylcholine to depolarize a skeletal muscle fiber to threshold
Explanation:
When an action potential reaches a neuromuscular junction, it causes acetylcholine to be released into this synapse. The acetylcholine binds to the nicotinic receptors concentrated on the motor end plate, a specialized area of the muscle fibre's post-synaptic membrane.
Acetylcholine is the neurotransmitter used at the neuromuscular junction—in other words, it is the chemical that motor neurons of the nervous system release in order to activate muscles. ... In the brain, acetylcholine functions as a neurotransmitter and as a neuromodulator.
Answer:
The correct answer will be option B- enzymatic action of Acetylcholinesterase.
Explanation:
At the neuromuscular junction, the neurotransmitter acetylcholine is released which then binds to the receptor in motor-end plate causes depolarization of the membrane responsible for the release of Ca⁺² ions. These Ca⁺² ions control the contraction of the muscle by binding to troponin.
This mechanism of muscle contraction is blocked after the breakdown of the acetylcholine by the "enzyme acetylcholinesterase" which leads to the closure of the Na⁺ ion-channels. The closure of these channels slows down the depolarization in muscle fiber and thus blocks the pathway which leads to muscle relaxation.
Thus, Option-B is the correct option.
