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ELEN [110]
3 years ago
8

which of the following scientific studies was made possible by the development of the light microscope

Biology
1 answer:
sergij07 [2.7K]3 years ago
6 0

Answer:

the study of cells as they are microscopic

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Fill in the blank with the appropriate term. 1.The___________ is often considered to be the cell's control center. 2.The________
ValentinkaMS [17]
 1.The nucleus is often considered to be the cell's control center.
(because it contains the DNA)

2.The cytoplasm consists of everything inside the plasma membrane of the cell.
(actually it excludes the nucleus)

 3.The plasma membrane forms a boundary between the inside and outside of the cell. - it controls what can enter and what can't!


4.The cytoskeleton is essentially a "skeleton" inside the cell..
it maintains the form of the cell

5.The rough endoplasmic reticulum is covered with
Ribosomes -they make the proteins!


6.Lysosomes use enzymes to break down foreign matter and dead cells.

 7.plant cells cells specifically have a cell wall, a large central vacuole, and chloroplasts.-choloplasts are only found in plants!

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3 years ago
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20 Points!!<br> Describe how an ionic compound is formed.
a_sh-v [17]

A metal and non-metal form into a compound. that compound is an ionic compound.

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3 years ago
Which method is best for seperating salt and breadcrumbs?
Tanzania [10]

Answer:

Filtering to obtains breadcrumbs and the heat the salt solution to saturation and cool it

8 0
2 years ago
WILL MARK BRAINLIEST!!
8090 [49]
The answer is B) Limiting factors.

Hope this Helps!!
4 0
3 years ago
Methylation of EZH2 by PRMT1 regulates its stability and promotes breast cancer metastasis. Cell Death Differ. 2020,
vovangra [49]

Yes, it is true that Methylation of EZH2 by PRMT1 regulates its stability and promotes breast cancer metastasis.

Enhancer of zeste homolog 2 (EZH2), a key histone methyltransferase and EMT inducer, is overexpressed in diverse carcinomas, including breast cancer.

However, the molecular mechanisms of EZH2 dysregulation in cancers are still largely unknown. Here, we discover that EZH2 is asymmetrically dimethylated at R342 (meR342-EZH2) by PRMT1.

meR342-EZH2 was found to inhibit the CDK1-mediated phosphorylation of EZH2 at T345 and T487, thereby attenuating EZH2 ubiquitylation mediated by the E3 ligase TRAF6.

We also demonstrate that meR342-EZH2 resulted in a decrease in EZH2 target gene expression, but an increase in breast cancer cell EMT, invasion and metastasis.

Moreover, we confirm the positive correlations among PRMT1, meR342-EZH2 and EZH2 expression in the breast cancer tissues. Finally, we report that high expression levels of meR342-EZH2 predict a poor clinical outcome in breast cancer patients.

Our findings may provide a novel diagnostic target and promising therapeutic target for breast cancer metastasis.

Learn more about breast cancer here : brainly.com/question/6747562

#SPJ4

4 0
2 years ago
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