Answer:
Repair mechanism for base cleavage (BER)
Explanation:
Repair by base cleavage (BER)
The altered bases are specifically recognized by glycosylases and removed, generating an AP site. The hole is filled by a DNA polymerase that takes the healthy strand as a template. This system arises not only by exposure to external agents, but also by the cell's own activity.
In case of damage in more than one nucleotide, repair by nucleotide excision (NER) is performed.
Nucleotide excision repair (NER)
The damaged area is recognized by UvrA and B, then A and B separate and UvrC enters which forms a complex with endonuclease activity with B. This enzyme cuts the T-dimer and the gap is filled by a DNA polymerase. There is also the TC-NER system (transcription-coupled nucleotide repair system). The alteration of these mechanisms gives rise to diseases such as: Xeroderma pigmentosum, Trichotiodystrophy or Cockayne Syndrome
Photosinthesis is where plants get their sunlight energy
Answer:
A) The gene flow between hatchery-reared and wild populations is leading to a decline in fitness of wild populations.
Explanation:
<u>we have relative fitness at its greatest in trouts whose parents are wild x wild. crossing the wild with hatchery reared causes a decrease in the fitness of the troutgene flow is the exchange of genes between two different breeds. the wild is a greatly diverse breed and it is highly adaptive. while the hatchery reared is less adaptive.when these two breed less fit alleles will then be transferred to the wild</u>
therefore option a answers the question
I think the correct answer from the choices listed above is option A. It is the alveoli that is composed of single layer <span>surrounded by a network of capillaries. These are used to allow oxygen and carbon dioxide to move from the lungs to the bloodstream.</span>
