The question is incomplete as it does not have the options which are:
photoreactivation
reactivation repair pathway
nucleotide excision repair pathway
UV repair pathway
p53 repair pathway
Answer:
p53 repair pathway
Explanation:
ATM gene or ataxia telangiectasia-mutated is a gene which codes for the protein kinase involved in the DNA double-stranded break.
The DNA double-stranded break is repaired easily as the protein kinase is recruited to the site of the break where it allows the DNA repair machinery to repair the DNA.
The gene also controls the cell growth therefore this gene is important to study cancer. Since the p53 protein is a suppressor protein which if mutated is repaired by the ATM gene and if the gene becomes mutated then the p53 repair pathway is affected.
Thus, p53 repair pathway is the correct answer.
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Both the sodium and the chlorine atoms need to obtain a stable electron configuration, thus, a complete outer shell of eight electrons.
Sodiums electron design is 2,8,1 and chlorines electron setup is 2,8,7. So all together for the two molecules of the two components to get a total external shell of eight electrons, sodium will give one electron to chlorine, and the chlorine particle will acknowledge the gave electron when responded with each other.
This outcomes in the sodium particle having an electron arrangement of 2,8 so is currently total. The chloride particle currently has an electron configuration of 2,8,8.
Since the molecules have moved toward becoming particles to end up stable, the Na+ and the Cl-particles draw in one another because of their contrary charges, which results in an ionic bond.
This is the reason sodium and chlorine cling to each other when responded.
