The small intestine receives food directly from the stomach and this is where food is mixed with strong digestive juices from the liver and pancreas
Answer:
A
Explanation:
Genetic mutations randomly occur in the population of cells. If, by chance, there is a mutation that gives the bacterial cell resistance to an antibiotic, then that mutation will remain in the population.
This is because it gives the cell a competitive advantage (because it is more likely to survive than its non-resistant counterparts). If it survives, it will divide and pass the mutation on to its daughter cells, which will survive. This keeps happening until an entire population will carry the mutation.
The correct answer is:
A. Will have a intermediate trait 3. Glucose aversion is a genetically-determined incompletely dominant trait
B. Will refuse glucose 2. Glucose aversion is a genetically-determined dominant trait
C. Have a mix of traits depending on experience 4. Glucose aversion is a learned behavior
D. Will accept glucose 1. Glucose aversion is a genetically-determined recessive trait
If we put it simple and say that for example glucose aversion is genetically determinated, with the genotype AA and eating glucose with the genotype aa. The offspring will have Aa genotype (heterozygous). Depending on which phenotype is expressed in heterozygous we can conclude whether the trait is dominant or recessive or due to earned behavior.
Answer: B voltage-gated potassium channels
Explanation:#
Tetraethylammonium is a pH- potassium channel blocker.
Therefore, it prevents the escape of potassium ions from the axoplasm, which makes it ( axoplam) positive from the rise in potassium ions concentration , since potassium ion is positive charged and intracellular ion . This initiate repolarization which returns to membrane state potential.
Since it is pH -potassium ion blocker; it does not affect the voltage-gated sodium ion channels, so action potential can occur. But after the sodium gated channels were closed,action potential drops but the concentration of potassium rises because :
1. They are naturally intracellular ions
2. They cannot leave the axoplasm because the channels has been shut. Therefore this stabilizes the membrane potential
I would say Cone cells are responsible for this
