Answer:
4. B cells become activated by interacting with helper T cells.
1. B cells display antigens in MHC class II receptors on the cell surface.
2. Antibodies released by plasma cells bind to the antigen so they will be destroyed by other cells of the immune system.
3.B cells rearrange their DNA to create a unique B-cell receptor.
5. B cells undergo clonal expansion.
6. B cells digest antigens that bind to the antibodies on their surface.
Explanation:
B-cells get activated by interacting with helper T cells when they bind to the antigen to receptors i.e (MHC class II receptors on the cell surface) on the surface of the cell. Series of activities such as release by plasma cells which cause rearrangement of B cells causes the cell to divide and proliferate. The process through which daughter cells arise from a parent cell called clonal expansion.
Answer:
According to the model, the "villi" of small intestines is being damaged as an impact of celiac disease. Small intestines are responsible for absorption of nutrients and minerals from food, this occurs through villi, in case of damaged villi absorption is interfered, thus disturbing the digestive system. If the nutrients, minerals or vitamins are not being absorbed well by small intestines then they will be excreted out of the body and will not be transported to the blood and other areas, this would result in deficiency of minerals and vitamins in blood that cause weakness.
The sperms and ovi has half the number of chromosomes of the somal cells, so they have 8.
The Krebs cycle can be used to make many various intermediates that can be siphoned off into other pathways. Mainly though, it makes the electron carriers NADH and FADH2 which can then be used in the electron transport chain to generate ATP.
