Answer:
No, it did not rain today in Texas.
Answer:
This outcome in a hybrid zone is known as <u><em>fusion</em></u>.
Explanation:
In the field of biology, hybrid zone can be described as the area where closely-related species can interbreed and produce hybrid organisms. Reinforcement can be described as the phenomenon by which a hybrid zone develops between full species barriers. Hybrid speciation can be described as the phenomenon by which a new species forms as the direct result of mating between members of two existing species.
With shorter necks, giraffe ancestors could not reach food-containing branches on tall trees. This resulted in the directional selection of giraffes with longer necks.
<h3>
Why is choosing a direction necessary? </h3>
It nearly appears obvious that the giraffe's long neck developed as a result of a lack of food in the lower branches of trees. The giraffe has a significant advantage because it is taller than any other mammal and can feed where few others can.
When compared to modern giraffes, the giraffe's ancient predecessors had a shorter neck. The plants that were lying at a higher level were inaccessible to them. Giraffe phenotypes have changed in various ways, and now have long necks to reach vegetation that is higher up. The extreme form is chosen above other features in directional selection. It was decided to choose the long-necked giraffe over the short-necked.
To know more about direction selection for longer necked giraffes visit:
brainly.com/question/3738222
#SPJ4
Answer:
shoots grow towards gravity
Answer:
a. p53 activates transcription of WAF1.
Explanation:
<u>WAF1 transcription occurs independent of p53 during oxidative stress so p53 does not play any role in cell cycle arrest in the signaling pathway which involves WAF1 . </u>
In rest of the mentioned options, p53 plays a role directly or indirectly. During double stranded lesion in DNA in G1 phase, a sensor protein known as ATM binds the DNA lesion site. ATM is a serine/threonine kinase which phosphorylates another kinase known as chk2. After phosphorylation,<u> chk2 stabilizes transcription factor p53.</u> p53 further acts as a transcription factor for the synthesis of a protein known as p21 which inhibits G1 phase specific CDK and ultimately cell is arrested in G1 phase. The cell remains in arrested state until the DNA lesion is fully repaired. <u>Hence, p53 indirectly blocks G1 to S transition with the help of p21. </u>
As such <u>WAF1 transcription factor involving pathway</u><u> </u><u>also requires p21 protein for causing cell cycle arrest but in this pathway p21 is not synthesized with the help of p53. </u>
