The body are digested by bacteria and thus recombines with soil and increase fertility of the soil !
Answer:
0.0177
Explanation:
Cystic fibrosis is an autosomal recessive disease, thereby an individual must have both copies of the CFTR mutant alleles to have this disease. The Hardy-Weinberg equilibrium states that p² + 2pq + q² = 1, where p² represents the frequency of the homo-zygous dominant genotype (normal phenotype), q² represents the frequency of the homo-zygous recessive genotype (cystic fibrosis phenotype), and 2pq represents the frequency of the heterozygous genotype (individuals that carry one copy of the CFTR mutant allele). Moreover, under Hardy-Weinberg equilibrium, the sum of the dominant 'p' allele frequency and the recessive 'q' allele frequency is equal to 1. In this case, we can observe that the frequency of the homo-zygous recessive condition for cystic fibrosis (q²) is 1/3200. In consequence, the frequency of the recessive allele for cystic fibrosis can be calculated as follows:
1/3200 = q² (have two CFTR mutant alleles) >>
q = √ (1/3200) = 1/56.57 >>
- Frequency of the CFTR allele q = 1/56.57 = 0.0177
- Frequency of the dominant 'normal' allele p = 1 - q = 1 - 0.0177 = 0.9823
Hormones glucagon and insulin are produced in the alpha and beta cells respectively in the Islet of Langerhan in the pancreas. They are involved in the negative feedback system of blood glucose regulation in homeostasis.
GLUCAGON: when there is a low blood glucose concentration, the pancreas detect this and alpha cells produce and release glucagon. Glucagon causes the cells of the body to absorb less glucose from the blood. It also inhibits the process of converting glucose into glycogen (glycogenesis) and cause gluconeogenesis (process of converting amino acids/proteins and lipids/fats into glucose) and glycogenolysis (conversion of glycogen to glucose). Finally, glucagon decreases the rate of respiration so less glucose is required.
INSULIN: when blood glucose is high, insulin is released. Insulin binds with cell surface receptors of cells and activates the enzymes attached to the receptor. The enzymes cause a conformational change in the structural proteins that surround glucose transport protein containing vesicles, causing them to move out of the way so the vesicles migrate up to the cell membrane and glucose transport proteins can fuse with it. Thus, more glucose can be taken in by cells. Insulin also cause glycogenesis (converting glucose into glycogen) and inhibits gluconeogenesis and glycogenolysis.
Basically insulin decreases blood glucose concentration (eg. after eating) and glucagon increases it (eg. skipping breakfast in the morning)
