Snapper overfishing
Explanation:
Coral reefs are becoming highly vulnerable due to increased anthropological exploitation of the ocean reserves. Overfishing, usage of non-selective fishing gears, fishing the spawning areas, fishing using nets etc all affect the coral reefs and the marine ecosystem. The coral reefs and the fish population are interdependent and overfishing greatly impacts both the factors.
Overfishing is the main reason for damage and decline in coral reefs. The snappers are carnivores and survive on eating other fishes which can be herbivores or omnivores. Snappers remain at the top of the food pyramid of the marine ecosystem. A reduction at the top level of the food pyramid will automatically disturb the lower level population and the energy flow between each cycle. A reduction in carnivores leads to increase in herbivores which in turn deplete the coral reefs. Coral reef is a habitat for many fish populations, and a reduction in the habitat will again reduce the overall population of fishes. A decrease in predator population will increase prey population.
Atoms tend to form bonds because one atom may have more attraction to electrons than the other
I think it's made up of 2 or more of the <em>same type</em> of atom.
A formed, photosynthesis, uses sunlight and carbon an uses it for energy or glucose
Thickening of gill epithelia in rainbow trout, caused by chloride cell proliferation, could lead to an impairment of oxygen uptake under moderate to severe hypoxia (Thomas et al 1988; Bindon et al., 1994; Greco et al., 1995).
<h3>What results in an increase in AMS in interstitial lung disease?</h3>
The number of alveolar macrophages (AMs) can rise in interstitial lung disease. Precursor cells from the peripheral circulation may have been drawn in, and/or there may have been local lung growth, to create this.
<h3>What connection does sarcoidosis have between lymphocytes and proliferating cells?</h3>
Additionally, a strong association between the quantities of lymphocytes and proliferative cells in sarcoidosis and fibrosis was discovered in bronchoalveolar lavage (BAL). Eosinophil counts and proliferating cell counts were positively associated in fibrosis.
<h3>How do AMS patients and healthy controls differ in terms of propagating AMS?</h3>
With a substantial association between these two indices, there was a considerable increase in proliferating AMs in all patient groups when compared to healthy controls (4.2 versus 1.4% Feulgen, and 2.1 against 0.5% Ki67).
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