Answer:
D. is caused by alcohol consumption
Explanation:
- ADH hormone is also called as vasopressin, it is secreted by the supraoptic nuclei of hypothalamus and released by posterior pituitary.
- The main function of ADH is to stimulate reabsorption of water from kidney tubules i.e DCT (distal convoluted tubules) and collecting duct.
- Anti diuretic means against diuresis (excess urination or loss of water). Insufficient ADH can lead to diabetes insipidus which includes the symptoms such as diuresis and dehydration but no glucose in urine.
- Alcohol suppresses the release of ADH, thus water reabsorption reduces and urine volume increases.
- Very high levels of ADH can result in dangerous conditions such as cerebral edema due to excess fluid buildup in the body.
- ADH allows the kidney to form concentrated urine while excreting normal amounts of salts. Continuous secretion of ADH may lead to hyponatremia i.e decrease of sodium levels in blood.
- Angiotensin II is released when there is low BP and it stimulates release of vasopressin/ADH to cause the vaso-constriction, there by bring the BP to normal.
Answer:
Prokaryotic cell:
Prokaryotic cell do not have the nucleus and their DNA is present in the cytoplasm. The membrane bound cell organelle are absent in the prokaryotic cells. The ribosome of prokaryotic cell is 70'S. Transcription and translation is coupled in prokaryotic cells.
Eukaryotic cells:
Eukaryotic cell have well defined nucleus and their DNA is present inside the nucleus. The membrane bound cell organelle are present in the eukaryotic cells. The ribosome of eukaryotic cell is 80'S. Transcription and translation is not coupled and takes place in separate compartments in eukaryotic cells.
Answer: False
Explanation: You're supposed to have numerically more smaller units than bigger units, since a lot of these smaller units are combined to create a single larger unit.
Answer:
b) opening calcium pores in the membrane
Explanation:
The opening of calcium ions channels facilitates the migration of vesicles containing neurotransmitters in the pre-synaptic neurons to the tip of the post-synaptic membrane. at the <u>neuromuscular junction </u>These neurotransmitters can either be <u>cholinergic or non-chorlinergic.</u> <u>At the tip of the pre-synaptic membranes the vesicles empty their neurotransmitter contents into the synapse.</u>
The neurotransmitters, diffuse through the neuromuscular junction to bind with the receptors on the <u>post-synaptic membrane on the post-synaptic neuron.</u> This stimulates opening of<u> ligand gated sodium ions,</u> and sodium ions diffuse into the post-synaptic neuronal membrane, to cause <u>depolarization</u>. if the stimulus is up to the threshold levels it generate action potential.
Therefore , more calcium ions channels opening produces; more diffusion of neurotransmitters;leading to opening of more sodium channels for depolarization; and IPSP or EPSP generation.
