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yan [13]
2 years ago
12

Describe how the care technique of adequate posture for bed ridden clients can impact parts of the respiratory system. Use corre

ct terminology of the respiratory system in your answer.
Medicine
1 answer:
romanna [79]2 years ago
6 0

Answer:

Patients who suffer from respiratory pathologies, generally present an increase in fluid in the alveoli, where they produce the gas exchange, that is why if we put the patient to bed completely, the gas exchange surface will be less, because the liquid that presents by the pulmonary emphysema is dispersed in more alveoli and therefore the difficulty of breathing is greater.

The ideal position then in this type of patient, where the problem is in the respiratory system, in the position of approximately 130 degrees, or an intermediate position between 180 and 90 degrees, since in this way the upper limb will not be at the same Height than the lower limb, the emphysema fluid does not disperse through the alveoli on a larger surface, but on a smaller surface, and thus the patient will be able to breathe better, and improve their gas exchange capacity.

Explanation:

The greater the occupied alveolar surface, the less capacity for gas exchange and therefore greater difficulty in breathing ... This would happen in patients who are fully reclined, that is, at 180 degrees.

If we position it well, between 90 and 180, approximately 130 degrees, less alveolar surface occupied by the fluid of the emphysema, greater gas exchange and therefore better breathing.

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Explain how neurons communicate. Include a description of the action potential and how the action potential is converted into a
suter [353]

Answer:

Action potentials and chemical neurotransmitters.

Explanation:

Neurons communicate with each other via electrical events called ‘action potentials’ and chemical neurotransmitters.  At the junction between two neurons (synapse), an action potential causes neuron A to release a chemical neurotransmitter.  The neurotransmitter can either help (excite) or hinder (inhibit) neuron B from firing its own action potential.

In an intact brain, the balance of hundreds of excitatory and inhibitory inputs to a neuron determines whether an action potential will result.  Neurons are essentially electrical devices. There are many channels sitting in the cell membrane (the boundary between a cell’s inside and outside) that allow positive or negative ions to flow into and out of the cell.  Normally, the inside of the cell is more negative than the outside; neuroscientists say that the inside is around -70 mV with respect to the outside, or that the cell’s resting membrane potential is -70 mV.

This membrane potential isn’t static. It’s constantly going up and down, depending mostly on the inputs coming from the axons of other neurons. Some inputs make the neuron’s membrane potential become more positive (or less negative, e.g. from -70 mV to -65 mV), and others do the opposite.

These are respectively termed excitatory and inhibitory inputs, as they promote or inhibit the generation of action potentials (the reason some inputs are excitatory and others inhibitory is that different types of neuron release different neurotransmitters; the neurotransmitter used by a neuron determines its effect).

Action potentials are the fundamental units of communication between neurons and occur when the sum total of all of the excitatory and inhibitory inputs makes the neuron’s membrane potential reach around -50 mV (see diagram), a value called the action potential threshold.  Neuroscientists often refer to action potentials as ‘spikes’, or say a neuron has ‘fired a spike’ or ‘spiked’. The term is a reference to the shape of an action potential as recorded using sensitive electrical equipment.

Neurons talk to each other across synapses. When an action potential reaches the presynaptic terminal, it causes neurotransmitter to be released from the neuron into the synaptic cleft, a 20–40nm gap between the presynaptic axon terminal and the postsynaptic dendrite (often a spine).

After travelling across the synaptic cleft, the transmitter will attach to neurotransmitter receptors on the postsynaptic side, and depending on the neurotransmitter released (which is dependent on the type of neuron releasing it), particular positive (e.g. Na+, K+, Ca+) or negative ions (e.g. Cl-) will travel through channels that span the membrane.

Synapses can be thought of as converting an electrical signal (the action potential) into a chemical signal in the form of neurotransmitter release, and then, upon binding of the transmitter to the postsynaptic receptor, switching the signal back again into an electrical form, as charged ions flow into or out of the postsynaptic neuron.

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3 years ago
Read this excerpt from Chapter 1 of / Know Why the Caged Bird Sings by Maya Angelou and answer the question.
DiKsa [7]

Answer:

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h5n1 avian influenza is widely considered to be the most serious global pandemic threat today and is the target of much worldwid
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Possible assortment of the influenza genome with swine and human strains

What is human h5n1 infections?

influenza virus strains that primarily affect birds but can also infect people.

The most common way to catch this disease is through contact with sick birds. Additionally, it can be spread from person to person.

Within two to eight days, symptoms start to appear, and they may resemble the common cold. Shortness of breath, muscle aches, headaches, fever, sore throat, and cough are possible symptoms.

In humans, the sickness may result in a high fatality rate. If administered within two days of symptoms, several antiviral medications may be helpful.

Types A, B, C, and D are the four different types of influenza viruses.

  • Humans and a variety of animals are both susceptible to influenza A viruses.
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  • Seals can contract infections, according to recent evidence.
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Hence, Possible assortment of the influenza genome with swine and human strains

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John stith pemberton was an american pharmacist who is best known as the inventor of what?.
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Answer:

John Stith Pemberton (July 8, 1831 – August 16, 1888) was an American pharmacist and Confederate States Army veteran who is best known as the inventor of Coca-Cola

Explanation:

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