Answer:
Nitrogenous bases contain the genetic information, their amount is variable among different species, and the arrangement of these bases is also variable among different species
Explanation:
Both Watson-Crick and Pauling's DNA models considered that DNA nitrogenous bases (i.e., Adenine, Cytosine, Thymine and Guanine) contain the genetic information that determines the characteristics of living organisms. Moreover, both DNA models also considered that nitrogenous base composition varies between species, as well as the arrangement of these bases in the DNA chain also varies between species. Based on these features, Linus Pauling considered that a model where nitrogenous bases would be arranged on the outside of the DNA molecule would be easier for the DNA molecule to be replicated, transcribed, or repaired. Although incorrect, Pauling's DNA triple helix model was fundamental to develop the helical (double-stranded) structure of DNA, which was finally discovered by Watson and Crick in 1953.
Answer:
b. Hydrogen-carbon
Explanation:
Triglycerides, also called fats are fatty acid esters of glycerol. One molecule of triglyceride consists of three fatty acids linked to one glycerol by ester linkages. Fatty acids are hydrocarbon derivatives with a long hydrocarbon chain in which carbon atoms are bonded to hydrogen atoms by covalent bonds. The carbon-hydrogen bond is one of the bonds with high energy levels. Therefore, the energy of triglycerides is stored in carbon-hydrogen bonds of hydrocarbon chains of fatty acids.
Answer:
True
Explanation:
A mutation is any alteration in the genetic sequence of the genome of a particular organism. Mutations in the germline (i.e., gametes) can pass to the next generation, thereby these mutations can increase their frequency in the population if they are beneficial or 'adaptive' for the organism in the environment in which the organism lives (in this case, an insect/bug). The mutation rate can be defined as the probability of mutations in a single gene/<em>locus</em>/organism over time. Mutation rates are highly variable and they depend on the organism/cell that suffers the mutation (e.g., prokaryotic cells are more prone to suffer mutations compared to eukaryotic cells), type of mutations (e.g., point mutations, fragment deletions, etc), type of genetic sequence (e.g., mitochondrial DNA sequences are more prone to suffer mutations compared to nuclear DNA), type of cell (multicellular organisms), stage of development, etc. Thus, the mutation rate is the frequency by which a genetic sequence changes from the wild-type to a 'mutant' variant, which is often indicated as the number of mutations <em>per</em> round of replication, <em>per</em> gamete, <em>per</em> cell division, etc. In a single gene sequence, the mutation rate can be estimated as the number of <em>de novo</em> mutations per nucleotide <em>per</em> generation. For example, in humans, the mutation rate ranges from 10⁻⁴ to 10⁻⁶ <em>per </em>gene <em>per</em> generation.