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natima [27]
3 years ago
7

I WILL MARK BRAINLIEST!!

Medicine
2 answers:
Fofino [41]3 years ago
7 0

Answer:

There are multiple reasons but two are..

Explanation:

1. The body has not been recovered

2. The body suffered transformation after death (ex. by fire or certain chemicals.. etc)

crimeas [40]3 years ago
7 0
1. The body has not been recovered

2. The body suffered transformation such as chemicals
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A cornea transplant involves the grafting of a donor cornea into a recipient’s anterior eye. The sutures to hold the graft in pl
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Answer: The cornea is avascular.

Explanation:

The cornea can be defined as the clear front membrane on the surface of eyeball. It lies in the front of the iris and pupil. It allows the light to enter the eye. A cornea transplant is recommended in patients who suffer from vision problems due to thinning of cornea, keratoconus, loss of vision and cloudiness of cornea.

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3 years ago
a client with an acute myocardial infarction is admitted to the coronary care unit. the health care provider has ordered heparin
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Myocardial infarction is Heart attack.

Myocardial infraction causes death and disability worldwide.

What is Myocardial infraction?

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6 0
2 years ago
Which statement about the prevention and treatment of TB is false?a) The BCG vaccine is important in reducing the transmission o
bekas [8.4K]

Answer:

hiv =ded

Explanation:

7 0
4 years ago
A dendritic cell is a(n)
brilliants [131]

Answer:

b) blastic red blood cell (RBC).

Explanation:

In excess of 340 blood group antigens have now been described that vary between individuals. Thus, any unit of blood that is nonautologous represents a significant dose of alloantigen. Most blood group antigens are proteins, which differ by a single amino acid between donors and recipients. Approximately 1 out of every 70 individuals are transfused each year (in the United States alone), which leads to antibody responses to red blood cell <u>(RBC) alloantigens</u> in some transfusion recipients. When alloantibodies are formed, in many cases, RBCs expressing the antigen in question can no longer be safely transfused. However, despite chronic transfusion, only 3% to 10% of recipients (in general) mount an alloantibody response. In some disease states, rates of alloimmunization are much higher (eg, sickle cell disease). For patients who become alloimmunized to multiple antigens, ongoing transfusion therapy becomes increasingly difficult or, in some cases, impossible. While alloantibodies are the ultimate immune effector of humoral alloimmunization, the cellular underpinnings of the immune system that lead to ultimate alloantibody production are complex, including antigen consumption, antigen processing, antigen presentation, T-cell biology.

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